Mitochondrial complex I deficiency leads to increased production of superoxide radicals and induction of superoxide dismutase.
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A ketogenic diet as a potential novel therapeutic intervention in amyotrophic lateral sclerosisMutant NDUFV2 subunit of mitochondrial complex I causes early onset hypertrophic cardiomyopathy and encephalopathyReplicative senescence induced by Romo1-derived reactive oxygen speciesA genomewide linkage-disequilibrium scan localizes the Saguenay-Lac-Saint-Jean cytochrome oxidase deficiency to 2p16The mechanism of superoxide production by NADH:ubiquinone oxidoreductase (complex I) from bovine heart mitochondriaGRACILE syndrome, a lethal metabolic disorder with iron overload, is caused by a point mutation in BCS1LOxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cellsSomatic mutations in mitochondria: the chicken or the egg?Somatic mutations in the mitochondria of rheumatoid arthritis synoviocytesAngiotensin II-superoxide-NFκB signaling and aortic baroreceptor dysfunction in chronic heart failureMitochondria-controlled signaling mechanisms of brain protection in hypoxiaDecoding Warburg's hypothesis: tumor-related mutations in the mitochondrial respiratory chainComplex I deficiency due to selective loss of Ndufs4 in the mouse heart results in severe hypertrophic cardiomyopathyRedox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cellsA novel syndrome affecting multiple mitochondrial functions, located by microcell-mediated transfer to chromosome 2p14-2p13.Mitochondrial alterations caused by defective peroxisomal biogenesis in a mouse model for Zellweger syndrome (PEX5 knockout mouse)Mitochondrial DNA mutations induce mitochondrial dysfunction, apoptosis and sarcopenia in skeletal muscle of mitochondrial DNA mutator miceVulnerability to oxidative stress in vitro in pathophysiology of mitochondrial short-chain acyl-CoA dehydrogenase deficiency: response to antioxidantsc-Myc and AMPK Control Cellular Energy Levels by Cooperatively Regulating Mitochondrial Structure and FunctionChanges in endoplasmic reticulum stress proteins and aldolase A in cells exposed to dopamineThe mitochondrial disease associated protein Ndufaf2 is dispensable for Complex-1 assembly but critical for the regulation of oxidative stressEndothelial mitochondria--less respiration, more integration.Investigation of the role of mitochondrial DNA in multiple sclerosis susceptibility.Pyrroloquinoline quinone stimulates mitochondrial biogenesis through cAMP response element-binding protein phosphorylation and increased PGC-1alpha expressionIdentification and functional clustering of global gene expression differences between human age-related cataract and clear lenses.Disruption of interleukin-1β autocrine signaling rescues complex I activity and improves ROS levels in immortalized epithelial cells with impaired cystic fibrosis transmembrane conductance regulator (CFTR) function.Degree of glutathione deficiency and redox imbalance depend on subtype of mitochondrial disease and clinical status.Role of mitochondria in mutant SOD1 linked amyotrophic lateral sclerosisDefects in mitochondrial axonal transport and membrane potential without increased reactive oxygen species production in a Drosophila model of Friedreich ataxiaThe nature and mechanism of superoxide production by the electron transport chain: Its relevance to aging.Mitochondria, oxygen free radicals, and apoptosis.Development of lipophilic cations as therapies for disorders due to mitochondrial dysfunction.Suppression of mitochondrial complex I influences cell metastatic properties.Aconitase causes iron toxicity in Drosophila pink1 mutantsAnalysis of mitochondrial ND1 gene in human colorectal cancerIncreased protein nitration in mitochondrial diseases: evidence for vessel wall involvement.ROS-triggered phosphorylation of complex II by Fgr kinase regulates cellular adaptation to fuel useDelivery of bioactive molecules to mitochondria in vivo.Mitochondrial encephalomyopathies.Complex I subunit gene therapy with NDUFA6 ameliorates neurodegeneration in EAE
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Mitochondrial complex I deficiency leads to increased production of superoxide radicals and induction of superoxide dismutase.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@en
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@nl
type
label
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@en
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@nl
prefLabel
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@en
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@nl
P2860
P356
P1476
Mitochondrial complex I defici ...... ction of superoxide dismutase.
@en
P2093
Pitkanen S
Robinson BH
P2860
P304
P356
10.1172/JCI118798
P407
P577
1996-07-01T00:00:00Z