Diminished post-rest potentiation of contractile force in human dilated cardiomyopathy. Functional evidence for alterations in intracellular Ca2+ handling.
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K201 improves aspects of the contractile performance of human failing myocardium via reduction in Ca2+ leak from the sarcoplasmic reticulumThe ryanodine receptor leak: how a tattered receptor plunges the failing heart into crisisMechanistic insight into the functional and toxic effects of Strophanthidin in the failing human myocardiumModelling sarcomeric cardiomyopathies in the dish: from human heart samples to iPSC cardiomyocytesSmall and large animal models in cardiac contraction research: advantages and disadvantagesFunctional Effects of Hyperthyroidism on Cardiac Papillary Muscle in Rats.Effects of biventricular pacing and scar size in a computational model of the failing heart with left bundle branch block.Axial stretch of rat single ventricular cardiomyocytes causes an acute and transient increase in Ca2+ spark rateRole of sodium-calcium exchanger in modulating the action potential of ventricular myocytes from normal and failing hearts.Ventricular dilation and electrical dyssynchrony synergistically increase regional mechanical nonuniformity but not mechanical dyssynchrony: a computational model.Tachycardia-induced diastolic dysfunction and resting tone in myocardium from patients with a normal ejection fractionExposure to phthalates affects calcium handling and intercellular connectivity of human stem cell-derived cardiomyocytesContractile strength during variable heart duration is species and preload dependent.A Spatially Detailed Model of Isometric Contraction Based on Competitive Binding of Troponin I Explains Cooperative Interactions between Tropomyosin and Crossbridges.Physiological response of cardiac tissue to bisphenol A: alterations in ventricular pressure and contractility.Store-operated Ca2+ entry supports contractile function in hearts of hibernators.The Frank-Starling mechanism involves deceleration of cross-bridge kinetics and is preserved in failing human right ventricular myocardium.Subcellular remodeling as a viable target for the treatment of congestive heart failure.Age and hypertrophy related changes in contractile post-rest behavior and action potential properties in isolated rat myocytes.Comparison between myocardial infarction and diabetes mellitus damage caused angiogenesis or energy metabolismRate-dependent force, intracellular calcium, and action potential voltage alternans are modulated by sarcomere length and heart failure induced-remodeling of thin filament regulation in human heart failure: A myocyte modeling study.Determinants of frequency-dependent contraction and relaxation of mammalian myocardium.Variability in interbeat duration influences myocardial contractility in rat cardiac trabeculae.Molecular basis of diastolic dysfunction.Cardiac resynchronization: insight from experimental and computational models.A random cycle length approach for assessment of myocardial contraction in isolated rabbit myocardium.Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart.Frequency-dependent contractile response of isolated cardiac trabeculae under hypo-, normo-, and hyperthermic conditions.Molecular characterization and functional properties of cardiomyocytes derived from human inducible pluripotent stem cells.Impaired sarcoplasmic reticulum function leads to contractile dysfunction and cardiac hypertrophy.Sarcoplasmic reticulum Ca(2+)-ATPase overexpression by adenovirus mediated gene transfer and in transgenic mice.Effects of deformation on transmural dispersion of repolarization using in silico models of human left ventricular wedge.An integrative appraisal of mechano-electric feedback mechanisms in the heart.Abnormal contraction caused by expression of G(i)-coupled receptor in transgenic model of dilated cardiomyopathy.Cardiac transgenesis with the tetracycline transactivator changes myocardial function and gene expression.Improvement of cardiomyocyte function by a novel pyrimidine-based CaMKII-inhibitor.Dynamic Model for Characterizing Contractile Behaviors and Mechanical Properties of a Cardiomyocyte.Reduction of SR Ca2+ leak and arrhythmogenic cellular correlates by SMP-114, a novel CaMKII inhibitor with oral bioavailability.Atrial myocardium is the predominant inotropic target of adrenomedullin in the human heart.Ca(2+) handling in isolated human atrial myocardium.
P2860
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P2860
Diminished post-rest potentiation of contractile force in human dilated cardiomyopathy. Functional evidence for alterations in intracellular Ca2+ handling.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@en
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@nl
type
label
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@en
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@nl
prefLabel
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@en
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@nl
P2093
P2860
P356
P1476
Diminished post-rest potentiat ...... n intracellular Ca2+ handling.
@en
P2093
Hasenfuss G
Holubarsch C
Olschewski M
Schmidt-Schweda S
Sütterlin M
P2860
P304
P356
10.1172/JCI118849
P407
P577
1996-08-01T00:00:00Z