Vascular permeability and pathological angiogenesis in caveolin-1-null mice. - Wikidata - awgldk - TriplyDB

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

http://www.wikidata.org/entity/Q37362134

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The Pathophysiology of Moyamoya Disease: An Update Tumors: wounds that do not heal-redux Angiopoietin-2-induced blood-brain barrier compromise and increased stroke size are rescued by VE-PTP-dependent restoration of Tie2 signaling.Vascular permeability--the essentials Registered report: Biomechanical remodeling of the microenvironment by stromal caveolin-1 favors tumor invasion and metastasis Vascular permeability to plasma, plasma proteins, and cells: an update.Ezetimibe suppresses cholesterol accumulation in lipid-loaded vascular smooth muscle cells in vitro via MAPK signaling A new role for the muscle repair protein dysferlin in endothelial cell adhesion and angiogenesis.Caveolae protect endothelial cells from membrane rupture during increased cardiac output Host deficiency in caveolin-2 inhibits lung carcinoma tumor growth by impairing tumor angiogenesis.Caveolae-dependent and -independent uptake of albumin in cultured rodent pulmonary endothelial cells.Orphan nuclear transcription factor TR3/Nur77 regulates microvessel permeability by targeting endothelial nitric oxide synthase and destabilizing endothelial junctions.Atrial natriuretic peptide enhances microvascular albumin permeability by the caveolae-mediated transcellular pathway.Biomechanical remodeling of the microenvironment by stromal caveolin-1 favors tumor invasion and metastasis Role of caveolin-2 in subcutaneous tumor growth and angiogenesis associated with syngeneic mouse Lewis lung carcinoma and B16 melanoma models Caveolae, caveolins, cavins, and endothelial cell function: new insights.Vascular hyperpermeability, angiogenesis, and stroma generation Genetic ablation of Cav1 differentially affects melanoma tumor growth and metastasis in mice: role of Cav1 in Shh heterotypic signaling and transendothelial migration.A Polymorphism in RNF213 Is a Susceptibility Gene for Intracranial Atherosclerosis.PV1 down-regulation via shRNA inhibits the growth of pancreatic adenocarcinoma xenografts.Loss of caveolin-1 alters extracellular matrix protein expression and ductal architecture in murine mammary glands Regulation of Cell Signaling and Function by Endothelial Caveolins: Implications in Disease Caveolin-1 deficiency decreases atherosclerosis by hampering leukocyte influx into the arterial wall and generating a regulatory T-cell response.Caveolin-1, Ring finger protein 213, and endothelial function in Moyamoya disease.Ultrasound and magnetic resonance molecular imaging of atherosclerotic neovasculature with perfluorocarbon magnetic nanocapsules targeted against vascular endothelial growth factor receptor 2 in rats.Catecholamines facilitate VEGF-dependent angiogenesis via β2-adrenoceptor-induced Epac1 and PKA activation.Caveolins and caveolae in ocular physiology and pathophysiology.Nucleoside diphosphate kinase B regulates angiogenic responses in the endothelium via caveolae formation and c-Src-mediated caveolin-1 phosphorylation.Caveolin-1 opens endothelial cell junctions by targeting catenins.Vascular endothelial growth factor increases permeability of the blood-tumor barrier via caveolae-mediated transcellular pathway.Loss of caveolin-1 causes blood-retinal barrier breakdown, venous enlargement, and mural cell alteration.Biological characteristics of foam cell formation in smooth muscle cells derived from bone marrow stem cells.Chromosome 7q31 POAG locus: ocular expression of caveolins and lack of association with POAG in a US cohort.Caveolin1 Is Required for Th1 Cell Infiltration, but Not Tight Junction Remodeling, at the Blood-Brain Barrier in Autoimmune Neuroinflammation.Scleroderma fibroblasts suppress angiogenesis via TGF-β/caveolin-1 dependent secretion of pigment epithelium-derived factor.Critical role of caveolin-1 in ocular neovascularization and multitargeted antiangiogenic effects of cavtratin via JNK.Altered angiogenesis in caveolin-1 gene-deficient mice is restored by ablation of endothelial nitric oxide synthase.Single-chain VEGF/Cy5.5 targeting vegf receptors to indicate atherosclerotic plaque instability.Computational Approach to Investigating Key GO Terms and KEGG Pathways Associated with CNV.Caveolin-1-Knockout Mouse as a Model of Inflammatory Diseases

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Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

http://www.wikidata.org/entity/Q37362134

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 03 September 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

@cs

name

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@en

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@nl

type

label

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@en

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@nl

prefLabel

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@en

Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

@nl

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AJPATH.2009.090171

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The American Journal of Pathology

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Vascular permeability and pathological angiogenesis in caveolin-1-null mice.

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Ann M Dvorak

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Dian Feng

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Harold F Dvorak

http://www.w3.org/2001/XMLSchema#string

Janice A Nagy

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Sung-Hee Chang

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Tracey E Sciuto

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P2860

The vesiculo-vacuolar organelle (VVO). A new endothelial cell permeability organelle

Caveolae and caveolins in the cardiovascular system

Molecular cloning of caveolin-3, a novel member of the caveolin gene family expressed predominantly in muscle

Caveolin-1 deficiency increases cerebral ischemic injury

Caveolin-1 knockout mice show an impaired angiogenic response to exogenous stimuli

Caveolin-1(-/-)- and caveolin-2(-/-)-deficient mice both display numerous skeletal muscle abnormalities, with tubular aggregate formation

Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice

Antitumor effects of in vivo caveolin gene delivery are associated with the inhibition of the proangiogenic and vasodilatory effects of nitric oxide.

Pathways of macromolecular extravasation across microvascular endothelium in response to VPF/VEGF and other vasoactive mediators.

Openings through endothelial cells associated with increased microvascular permeability.

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scholarly article

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10.2353/AJPATH.2009.090171

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2009-09-03T00:00:00Z

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19729487

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2751571

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