Irreversibility of cellular aging and neoplastic transformation: a clonal analysis. - Wikidata - awgldk - TriplyDB

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

http://www.wikidata.org/entity/Q37363643

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Ubiquitous, heritable damage in cell populations that survive treatment with methotrexate.SSeCKS, a major protein kinase C substrate with tumor suppressor activity, regulates G(1)-->S progression by controlling the expression and cellular compartmentalization of cyclin D.

P2860

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P2860

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

http://www.wikidata.org/entity/Q37363643

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on September 1996

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@en

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@nl

type

label

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@en

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@nl

prefLabel

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@en

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@nl

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PNAS.93.18.9793

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Irreversibility of cellular aging and neoplastic transformation: a clonal analysis.

@en

P2093

Chow M

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Rubin H

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P2860

THE LIMITED IN VITRO LIFETIME OF HUMAN DIPLOID CELL STRAINS

Neoplastic development: paradoxical relation between impaired cell growth at low population density and excessive growth at high density

Physiological induction and reversal of focus formation and tumorigenicity in NIH 3T3 cells.

Etiological factors, stages, and the role of the foreign body in foreign body tumorigenesis: a review.

Colony size distributions as a measure of in vivo and in vitro aging.

Age and the cell cycle of the mouse esophageal epithelium.

Heritable, population-wide damage to cells as the driving force of neoplastic transformation.

Decreased fidelity of DNA polymerase activity isolated from aging human fibroblasts

The relationship between in vitro cellular aging and in vivo human age.

Gene amplification; what are we learning?

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9793-9798

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scholarly article

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10.1073/PNAS.93.18.9793

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18

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1996-09-01T00:00:00Z

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14413434

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8790410

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