Cell cycle inhibition preserves endothelial function in genetically engineered rabbit vein grafts. - Wikidata - awgldk - TriplyDB

Cell cycle inhibition preserves endothelial function in genetically engineered rabbit vein grafts.

Cell cycle inhibition preserves endothelial function in genetically engineered rabbit vein grafts.

http://www.wikidata.org/entity/Q37365179

about

Improved gene delivery to human saphenous vein cells and tissue using a peptide-modified adenoviral vector Gene Therapy in Cardiac Surgery: Clinical Trials, Challenges, and Perspectives Molecular advances in cardiac and cardiovascular disease.Therapeutic applications of transcription factor decoy oligonucleotides Targeting the cell cycle machinery for the treatment of cardiovascular disease.Inhibition of transforming growth factor-β restores endothelial thromboresistance in vein grafts.Degenerative valve disease and bioprostheses: risk assessment, predictive diagnosis, personalised treatments Thiram inhibits angiogenesis and slows the development of experimental tumours in mice.Gene therapy for the prevention of vein graft disease Gene therapy to prevent occlusion of venous bypass grafts.Pressure-mediated oligonucleotide transfection of rat and human cardiovascular tissues.Coronary artery bypass graft: why is the saphenous vein prone to intimal hyperplasia?New therapeutic possibilities for vein graft disease in the post-edifoligide era.Oligonucleotide-based gene therapy for cardiovascular disease.Regulation of vascular smooth muscle cell growth: targeting the final common pathway New prospects for cardiovascular gene therapy.Reduced viability of human vascular endothelial cells cultured on Matrigel.

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P2860

Cell cycle inhibition preserves endothelial function in genetically engineered rabbit vein grafts.

http://www.wikidata.org/entity/Q37365179

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on March 1997

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@en

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@nl

type

label

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@en

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@nl

prefLabel

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@en

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@nl

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P1433

Journal of Clinical Investigation

P1476

Cell cycle inhibition preserve ...... engineered rabbit vein grafts.

@en

P2093

G H Gibbons

http://www.w3.org/2001/XMLSchema#string

H E von der Leyen

http://www.w3.org/2001/XMLSchema#string

J P Cooke

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M J Mann

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P S Tsao

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R Buitrago

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R Kernoff

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V J Dzau

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P2860

Vascular cell adhesion molecule-1 (VCAM-1) gene transcription and expression are regulated through an antioxidant-sensitive mechanism in human vascular endothelial cells.

Evidence for enhanced vascular superoxide anion production in nitrate tolerance. A novel mechanism underlying tolerance and cross-tolerance.

Proliferating or interleukin 1-activated human vascular smooth muscle cells secrete copious interleukin 6.

Genetic engineering of vein grafts resistant to atherosclerosis

Impaired muscarinic endothelium-dependent relaxation and cyclic guanosine 5'-monophosphate formation in atherosclerotic human coronary artery and rabbit aorta.

Gene therapy inhibiting neointimal vascular lesion: in vivo transfer of endothelial cell nitric oxide synthase gene.

Modified low density lipoprotein and its constituents augment cytokine-activated vascular cell adhesion molecule-1 gene expression in human vascular endothelial cells.

Cytokines and growth factors in atherogenesis.

Hypercholesterolemia increases endothelial superoxide anion production

Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines.

P304

1295-1301

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10.1172/JCI119288

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P407

P433

6

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99

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1997-03-01T00:00:00Z

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507945

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