TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
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Roles of adaptor proteins in podocyte biologyRole of the TGF-β/BMP-7/Smad pathways in renal diseasesCD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survivalMpv17 in mitochondria protects podocytes against mitochondrial dysfunction and apoptosis in vivo and in vitro.BAMBI is expressed in endothelial cells and is regulated by lysosomal/autolysosomal degradationTranscriptional activation of the human CD2AP promoter by E2F1.Smad2-dependent downregulation of miR-30 is required for TGF-β-induced apoptosis in podocytesMicroRNA-21 in glomerular injury.Qiwei granules alleviates podocyte lesion in kidney of diabetic KK-Ay mice.Influence of tacrolimus on podocyte injury inducted by angiotensin II.Mechanisms and consequences of TGF-ß overexpression by podocytes in progressive podocyte disease.Pentoxifylline Attenuates Proteinuria in Anti-Thy1 Glomerulonephritis via Downregulation of Nuclear Factor-κB and Smad2/3 Signaling.Transforming growth factor-β, bioenergetics, and mitochondria in renal diseaseTransforming Growth Factor β1-induced Apoptosis in Podocytes via the Extracellular Signal-regulated Kinase-Mammalian Target of Rapamycin Complex 1-NADPH Oxidase 4 AxisDeleting the TGF-β receptor attenuates acute proximal tubule injury.TGF-β1 stimulates mitochondrial oxidative phosphorylation and generation of reactive oxygen species in cultured mouse podocytes, mediated in part by the mTOR pathway.Molecular basis of etiological implications in Alzheimer's disease: focus on neuroinflammation.Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.Expression of TGF-β1/mTOR signaling pathway in pathological scar fibroblasts.CIN85/RukL is a novel binding partner of nephrin and podocin and mediates slit diaphragm turnover in podocytesInhibition of SHIP2 in CD2AP-deficient podocytes ameliorates reactive oxygen species generation but aggravates apoptosisDexamethasone inhibits podocyte apoptosis by stabilizing the PI3K/Akt signal pathway.Astragaloside effect on TGF-β1, SMAD2/3, and α-SMA expression in the kidney tissues of diabetic KKAy mice.Triptolide protects podocytes from TGF-β-induced injury by preventing miR-30 downregulation.Transforming growth factor-β1 and phosphatases modulate COX-2 protein expression and TAU phosphorylation in cultured immortalized podocytes.The Role of Trio, a Rho Guanine Nucleotide Exchange Factor, in Glomerular Podocytes.TGFβ-Induced Actin Cytoskeleton Rearrangement in Podocytes Is Associated with Compensatory Adaptation of Mitochondrial Energy Metabolism.Upregulation of mitochondrial Nox4 mediates TGF-β-induced apoptosis in cultured mouse podocytes.Diabetic conditions downregulate the expression of CD2AP in podocytes via PI3-K/Akt signalling.
P2860
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P2860
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 13 August 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
@en
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes.
@nl
type
label
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
@en
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes.
@nl
prefLabel
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
@en
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes.
@nl
P2093
P2860
P356
P1476
TbetaRI independently activates Smad- and CD2AP-dependent pathways in podocytes
@en
P2093
Andrey S Shaw
Erwin P Böttinger
Sandhya Xavier
Stefanie Krick
Taoran Zhang
Thiruvur Niranjan
P2860
P304
P356
10.1681/ASN.2008070806
P577
2009-08-13T00:00:00Z