DNA damage-induced sustained p53 activation contributes to inflammation-associated hepatocarcinogenesis in rats.
about
Current and emerging biomarkers of cell death in human disease.Low-dose ionizing irradiation triggers a 53BP1 response to DNA double strand breaks in mouse spermatogonial stem cells.p53 amplifies Toll-like receptor 5 response in human primary and cancer cells through interaction with multiple signal transduction pathwaysp53 promotes inflammation-associated hepatocarcinogenesis by inducing HMGB1 release.Absence of a p53 allele delays nitrogen mustard-induced early apoptosis and inflammation of murine skinThe matricellular protein CCN1 suppresses hepatocarcinogenesis by inhibiting compensatory proliferation.p53 as a Dichotomous Regulator of Liver Disease: The Dose Makes the Medicine.Diethylnitrosamine Increases Proliferation in Early Stages of Hepatic Carcinogenesis in Insulin-Treated Type 1 Diabetic Mice.
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DNA damage-induced sustained p53 activation contributes to inflammation-associated hepatocarcinogenesis in rats.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 October 2012
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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type
label
DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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prefLabel
DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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DNA damage-induced sustained p ...... hepatocarcinogenesis in rats.
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10.1038/ONC.2012.451
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2012-10-15T00:00:00Z