Persistent pain is dependent on spinal mitochondrial antioxidant levels
about
Use of Capsaicin to Treat Pain: Mechanistic and Therapeutic ConsiderationsNeuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms?P2X7 Cell Death Receptor Activation and Mitochondrial Impairment in Oxaliplatin-Induced Apoptosis and Neuronal Injury: Cellular Mechanisms and In Vivo ApproachA Wnt5a signaling pathway in the pathogenesis of HIV-1 gp120-induced painInnovations in the Management of Musculoskeletal Pain With Alpha-Lipoic Acid (IMPALA Trial): Study protocol for a Double-Blind, Randomized, Placebo-Controlled Crossover Trial of Alpha-Lipoic Acid for the Treatment of Fibromyalgia PainInvolvement of reactive oxygen species in long-term potentiation in the spinal cord dorsal horn.Supraspinal inactivation of mitochondrial superoxide dismutase is a source of peroxynitrite in the development of morphine antinociceptive tolerance.Superoxide and Nitric Oxide Involvement in Enhancing of N-methyl-D-aspartate Receptor-Mediated Central Sensitization in the Chronic Post-ischemia Pain ModelReactive oxygen species are involved in group I mGluR-mediated facilitation of nociceptive processing in amygdala neurons.Global inhibition of reactive oxygen species (ROS) inhibits paclitaxel-induced painful peripheral neuropathy.Spinal NADPH oxidase is a source of superoxide in the development of morphine-induced hyperalgesia and antinociceptive toleranceAdditive antinociceptive effects of a combination of vitamin C and vitamin E after peripheral nerve injury.Oxidant-induced activation of cGMP-dependent protein kinase Iα mediates neuropathic pain after peripheral nerve injury.TRPA1: a transducer and amplifier of pain and inflammation.The connection of monocytes and reactive oxygen species in pain.Mitochondrial reactive oxygen species are activated by mGluR5 through IP3 and activate ERK and PKA to increase excitability of amygdala neurons and pain behavior.Induction of long-term potentiation and long-term depression is cell-type specific in the spinal cord.Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.Reactive oxygen species mediate visceral pain-related amygdala plasticity and behaviors.Reactive Oxygen Species Donors Increase the Responsiveness of Dorsal Horn Neurons and Induce Mechanical Hyperalgesia in Rats.Prooxidant-induced c-Src/nuclear factor kappa B-coupled signalling in sensory ganglia mediates cutaneous hyperalgesia.HSV vector-mediated GAD67 suppresses neuropathic pain induced by perineural HIV gp120 in rats through inhibition of ROS and Wnt5aWingless-type mammary tumor virus integration site family, member 5A (Wnt5a) regulates human immunodeficiency virus type 1 (HIV-1) envelope glycoprotein 120 (gp120)-induced expression of pro-inflammatory cytokines via the Ca2+/calmodulin-dependent pSpinal mitochondrial-derived peroxynitrite enhances neuroimmune activation during morphine hyperalgesia and antinociceptive tolerance.Vitamin e, an antioxidant, as a possible therapeutic agent for treating painThe Molecular and Pharmacological Mechanisms of HIV-Related Neuropathic Pain.Acupuncture and Traditional Chinese Medicine for the management of a 35-year-old man with chronic prostatitis with chronic pelvic pain syndrome.Spinal astrocytic activation contributes to mechanical allodynia in a rat model of cyclophosphamide-induced cystitis.Distinct contributions of reactive oxygen species in amygdala to bee venom-induced spontaneous pain-related behaviorsExtracellular caspase-6 drives murine inflammatory pain via microglial TNF-α secretionα-Lipoic acid and superoxide dismutase in the management of chronic neck pain: a prospective randomized study.Reactive nitroxidative species and nociceptive processing: determining the roles for nitric oxide, superoxide, and peroxynitrite in pain.Roles of reactive oxygen and nitrogen species in painThe role of mitochondrial dysfunctions due to oxidative and nitrosative stress in the chronic pain or chronic fatigue syndromes and fibromyalgia patients: peripheral and central mechanisms as therapeutic targets?Understanding the role of mitochondria in the pathogenesis of chronic pain.Reactive oxygen species affect spinal cell type-specific synaptic plasticity in a model of neuropathic pain.Activation of NLRP3 inflammasome in peripheral nerve contributes to paclitaxel-induced neuropathic pain.Nitroxidative Signaling Mechanisms in Pathological Pain.NMDA-receptor activation and nitroxidative regulation of the glutamatergic pathway during nociceptive processing.MnSOD mediated by HSV vectors in the periaqueductal gray suppresses morphine withdrawal in rats.
P2860
Q28073741-0BE7B871-24CF-4F2C-99C9-F71E9E83588EQ28083464-2EE7A1C2-1779-4748-A328-06262FA0999AQ28534383-13463D30-BA77-4B5C-9037-90EAEE856A9DQ28604010-FBE92B7D-2488-4A71-B83B-17C4B499D267Q33555352-011F398F-B8DC-4F34-80C5-92F8B51CC163Q33593550-D2BE4045-DB6C-48EF-9DDE-09A2FF4D37D4Q33813707-409A5132-E034-4FCE-81FC-506D18F28428Q33908218-6F8DC694-22B0-4EF1-9B50-32290D52910BQ33995347-606B3243-0E67-4998-BD8F-F50F3DA6AA69Q34038073-8D3EDBC2-97F1-459B-ACE4-FB5498ADDABCQ34103705-36287ADE-522A-4B5E-A0D5-D4235A8E3D36Q34110860-A5CD19FF-A99A-4957-82BA-EB61B3CD4BC2Q34154543-3B762AB9-EDA5-48C1-BD6A-2B3BBBDBE333Q34376133-7E76643F-27C4-42BA-B53C-02A74C4AC16AQ34713303-D94C7ECA-6D0F-4A84-ADFC-19FBA44698AEQ34776360-165A537F-F1B5-4BE5-9632-B02E32B9FB6BQ35193964-FDD98905-DBB9-4756-9F71-16D95D0707A2Q35229769-83B9B2A8-BAD1-492F-AD3F-7310AD2EB612Q35472078-598EFA27-CE3D-442C-816A-7561C42EF3EFQ36118999-1A655E1F-E8D1-4774-9F1E-ED28B5F2495FQ36751546-2FE1FDFA-4971-4861-BD59-F14359A4BD90Q36777541-F254057B-D783-472D-AD9A-559554933D34Q36832652-795F26E9-EBA3-4EEB-ABAE-CCB38D437CA8Q36920496-EBFF8980-8263-4865-9638-6F0AB4398349Q37011098-7F57A1C9-0449-4A7C-B49C-5D82D8BC2164Q37149019-CED8789C-0C88-4BCA-A843-0E621267867DQ37337934-03B823AA-3FBE-429F-8122-F139C063768FQ37426010-1558A7A6-8DA6-4D3F-9525-154C16A9CA4BQ37446109-1A1CDA72-524D-4DC3-8A89-D64FE2407726Q37602200-F1DBEEA9-119F-4E51-895B-527904DFA42BQ37660456-41511DBA-2689-4A41-BB45-26175697C173Q37765512-DB445CDA-B9C6-4DE5-8E0E-C14DFB83C0F7Q37833285-977A069B-C41D-4367-83D9-F22A390B042DQ38120080-9D8197A3-2EFE-4AEA-8302-209F45DC79ADQ38154874-375917A9-5CDA-4C0F-83DF-96272F6DCC68Q38678930-07FB1FBC-E20A-4418-BB85-A7D1F0797589Q38697158-EFB5ABEE-81DB-4DD0-972E-9019035F210BQ39008516-F36E1628-656F-454A-936B-5A57B5AC3B35Q39603918-1E87E706-C62B-4D35-BCE0-46A5A79FD530Q40262488-FDD61D3F-A404-4551-8507-4F1E86CD937E
P2860
Persistent pain is dependent on spinal mitochondrial antioxidant levels
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Persistent pain is dependent on spinal mitochondrial antioxidant levels
@en
Persistent pain is dependent on spinal mitochondrial antioxidant levels.
@nl
type
label
Persistent pain is dependent on spinal mitochondrial antioxidant levels
@en
Persistent pain is dependent on spinal mitochondrial antioxidant levels.
@nl
prefLabel
Persistent pain is dependent on spinal mitochondrial antioxidant levels
@en
Persistent pain is dependent on spinal mitochondrial antioxidant levels.
@nl
P2093
P2860
P1476
Persistent pain is dependent on spinal mitochondrial antioxidant levels
@en
P2093
Eric Klann
Erica S Schwartz
Hee Young Kim
Inhyung Lee
Jigong Wang
Jin Mo Chung
Kyungsoon Chung
P2860
P304
P356
10.1523/JNEUROSCI.3792-08.2009
P407
P577
2009-01-01T00:00:00Z