about
TRB3 links the E3 ubiquitin ligase COP1 to lipid metabolismSUMO modification regulates the transcriptional activity of XBP1Angptl4 protects against severe proinflammatory effects of saturated fat by inhibiting fatty acid uptake into mesenteric lymph node macrophagesDeficiency of suppressor enhancer Lin12 1 like (SEL1L) in mice leads to systemic endoplasmic reticulum stress and embryonic lethalityThe CREB coactivator TORC2 is a key regulator of fasting glucose metabolismThe ER-associated degradation adaptor protein Sel1L regulates LPL secretion and lipid metabolismA Phos-tag-based approach reveals the extent of physiological endoplasmic reticulum stress.Mechanisms of inflammatory responses in obese adipose tissue.Diet-induced alterations in gut microflora contribute to lethal pulmonary damage in TLR2/TLR4-deficient mice.The roles of ATF3, an adaptive-response gene, in high-fat-diet-induced diabetes and pancreatic beta-cell dysfunction.The pseudokinase tribbles homolog 3 interacts with ATF4 to negatively regulate insulin exocytosis in human and mouse beta cells.XBP-1 couples endoplasmic reticulum stress to augmented IFN-beta induction via a cis-acting enhancer in macrophages.Overexpression of TRB3 in muscle alters muscle fiber type and improves exercise capacity in mice.A CRISPR-Based Screen Identifies Genes Essential for West-Nile-Virus-Induced Cell Death.The full capacity of AICAR to reduce obesity-induced inflammation and insulin resistance requires myeloid SIRT1Short telomeres and ataxia-telangiectasia mutated deficiency cooperatively increase telomere dysfunction and suppress tumorigenesis.Telomere fusion to chromosome breaks reduces oncogenic translocations and tumour formation.Chronic intake of high fish oil diet induces myeloid-derived suppressor cells to promote tumor growth.The transcriptional co-regulator HCF-1 is required for INS-1 β-cell glucose-stimulated insulin secretionGr-1+ CD11b+ myeloid-derived suppressor cells suppress inflammation and promote insulin sensitivity in obesity.A conserved structural determinant located at the interdomain region of mammalian inositol-requiring enzyme 1alphaHigh-Resolution Metabolomics with Acyl-CoA Profiling Reveals Widespread Remodeling in Response to Diet.Epithelial Sel1L is required for the maintenance of intestinal homeostasisFish Oil-Rich Diet Promotes Hematopoiesis and Alters Hematopoietic Niche.Activation of natural killer T cells promotes M2 Macrophage polarization in adipose tissue and improves systemic glucose tolerance via interleukin-4 (IL-4)/STAT6 protein signaling axis in obesity.The ATP-P2X7 signaling axis is dispensable for obesity-associated inflammasome activation in adipose tissue.Short term high fat diet challenge promotes alternative macrophage polarization in adipose tissue via natural killer T cells and interleukin-4.IRE1α is an endogenous substrate of endoplasmic-reticulum-associated degradationDirect control of hepatic glucose production by interleukin-13 in mice.Nonmuscle myosin IIB links cytoskeleton to IRE1α signaling during ER stress.Phenformin activates the unfolded protein response in an AMP-activated protein kinase (AMPK)-dependent manner.Sel1L is indispensable for mammalian endoplasmic reticulum-associated degradation, endoplasmic reticulum homeostasis, and survival.Adipocyte spliced form of X-box-binding protein 1 promotes adiponectin multimerization and systemic glucose homeostasis.Tipping the balance in metabolic regulation: regulating regulatory T cells by costimulationEmerging roles for XBP1, a sUPeR transcription factor.Stressed out about obesity: IRE1α-XBP1 in metabolic disorders.Endoplasmic reticulum quality control in cancer: Friend or foe.Hypoxia-inducible lipid droplet-associated (HILPDA) is not a direct physiological regulator of lipolysis in adipose tissue.Developing robust, hydrogel-based, nanofiber-enabled encapsulation devices (NEEDs) for cell therapies.Hypoxia-inducible lipid droplet-associated (HILPDA) is a novel peroxisome proliferator-activated receptor (PPAR) target involved in hepatic triglyceride secretion.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Ling Qi
@ast
Ling Qi
@en
Ling Qi
@es
Ling Qi
@nl
Ling Qi
@sl
type
label
Ling Qi
@ast
Ling Qi
@en
Ling Qi
@es
Ling Qi
@nl
Ling Qi
@sl
prefLabel
Ling Qi
@ast
Ling Qi
@en
Ling Qi
@es
Ling Qi
@nl
Ling Qi
@sl
P106
P31
P496
0000-0001-8229-0184
P569
2000-01-01T00:00:00Z