Cutting edge: contribution of lung-resident T cell proliferation to the overall magnitude of the antigen-specific CD8 T cell response in the lungs following murine influenza virus infection.
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Balancing Immune Protection and Immune Pathology by CD8(+) T-Cell Responses to Influenza InfectionThe Timing of T Cell Priming and CyclingHow Neutrophils Shape Adaptive Immune ResponsesThe Role of Invariant Natural Killer T Cells in Dendritic Cell Licensing, Cross-Priming, and Memory CD8(+) T Cell GenerationRegulating the adaptive immune response to respiratory virus infectionDendritic cells and influenza A virus infectionImmune suppression by neutrophils and granulocytic myeloid-derived suppressor cells: similarities and differencesThree phases of CD8 T cell response in the lung following H1N1 influenza infection and sphingosine 1 phosphate agonist therapyCross-reactive T cells are involved in rapid clearance of 2009 pandemic H1N1 influenza virus in nonhuman primatesA pandemic influenza H1N1 live vaccine based on modified vaccinia Ankara is highly immunogenic and protects mice in active and passive immunizationsThe effector T cell response to influenza infectionSwine influenza H1N1 virus induces acute inflammatory immune responses in pig lungs: a potential animal model for human H1N1 influenza virus.Sequencing, annotation, and characterization of the influenza ferret infectome.The role of T-cell receptor recognition of peptide:MHC complexes in the formation and activity of Foxp3⁺ regulatory T cells.IL-15 trans-presentation by pulmonary dendritic cells promotes effector CD8 T cell survival during influenza virus infection.Every breath you take: the impact of environment on resident memory CD8 T cells in the lung.Persistence in Temporary Lung Niches: A Survival Strategy of Lung-Resident Memory CD8+ T Cells.Rapid reactivation of extralymphoid CD4 T cells during secondary infectionEvasion of innate and adaptive immune responses by influenza A virus.Dissolving polymer microneedle patches for influenza vaccination.Memory T cells persisting within the brain after local infection show functional adaptations to their tissue of residenceVaginal type-II mucosa is an inductive site for primary CD8⁺ T-cell mucosal immunity.Inflammatory chemokine receptors regulate CD8(+) T cell contraction and memory generation following infectionThe magnitude of the T cell response to a clinically significant dose of influenza virus is regulated by TRAIL.Modeling of influenza-specific CD8+ T cells during the primary response indicates that the spleen is a major source of effectors.Absence of Stat1 in donor CD4⁺ T cells promotes the expansion of Tregs and reduces graft-versus-host disease in miceChronic ethanol exposure selectively inhibits the influenza-specific CD8 T cell response during influenza a virus infection.Influenza Infection in Mice Induces Accumulation of Lung Mast Cells through the Recruitment and Maturation of Mast Cell ProgenitorsTSLP promotes influenza-specific CD8+ T-cell responses by augmenting local inflammatory dendritic cell functionPlasmacytoid Dendritic Cells Require Direct Infection To Sustain the Pulmonary Influenza A Virus-Specific CD8 T Cell Response.Protective CD8 T cell-mediated immunity against influenza A virus infection following influenza virus-like particle vaccination.Tumor necrosis factor receptor/tumor necrosis factor family members in antiviral CD8 T-cell immunity.CD8(+) T cells: foot soldiers of the immune systemOptimizing vaccine-induced CD8(+) T-cell immunity: focus on recombinant adenovirus vectors.OX40:OX40L axis: emerging targets for improving poxvirus-based CD8(+) T-cell vaccines against respiratory virusesTCR signaling requirements for activating T cells and for generating memory.Effector T-cell responses in non-lymphoid tissues: insights from in vivo imaging.Immunological assessment of influenza vaccines and immune correlates of protection.Peripheral regulation of T cells by dendritic cells during infection.Dynamics of influenza-induced lung-resident memory T cells underlie waning heterosubtypic immunity.
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P2860
Cutting edge: contribution of lung-resident T cell proliferation to the overall magnitude of the antigen-specific CD8 T cell response in the lungs following murine influenza virus infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Cutting edge: contribution of ...... ine influenza virus infection.
@en
Cutting edge: contribution of ...... ine influenza virus infection.
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type
label
Cutting edge: contribution of ...... ine influenza virus infection.
@en
Cutting edge: contribution of ...... ine influenza virus infection.
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prefLabel
Cutting edge: contribution of ...... ine influenza virus infection.
@en
Cutting edge: contribution of ...... ine influenza virus infection.
@nl
P2860
P356
P1476
Cutting edge: contribution of ...... ine influenza virus infection.
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P2093
Jodi McGill
P2860
P304
P356
10.4049/JIMMUNOL.0901109
P407
P577
2009-10-01T00:00:00Z