Learning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels? - Wikidata - awgldk - TriplyDB

Learning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels?

Learning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels?

http://www.wikidata.org/entity/Q37392258

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Arsenic Exposure Induces Unscheduled Mitotic S Phase Entry Coupled with Cell Death in Mouse Cortical Astrocytes Effects of low-dose drinking water arsenic on mouse fetal and postnatal growth and development In utero and early life arsenic exposure in relation to long-term health and disease A cross-sectional study of well water arsenic and child IQ in Maine schoolchildren Learning about cognition risk with the radial-arm maze in the developmental neurotoxicology battery Effects of prenatal exposure to sodium arsenite on motor and food-motivated behaviors from birth to adulthood in C57BL6/J mice.Fluoxetine treatment ameliorates depression induced by perinatal arsenic exposure via a neurogenic mechanism Reference compounds for alternative test methods to indicate developmental neurotoxicity (DNT) potential of chemicals: example lists and criteria for their selection and use.Adult hippocampal neurogenesis and mRNA expression are altered by perinatal arsenic exposure in mice and restored by brief exposure to enrichment.Regional specific groundwater arsenic levels and neuropsychological functioning: a cross-sectional study.Genome-wide alteration of histone H3K9 acetylation pattern in mouse offspring prenatally exposed to arsenic Long-term low-level arsenic exposure is associated with poorer neuropsychological functioning: a Project FRONTIER study.Ablation of Fmrp in adult neural stem cells disrupts hippocampus-dependent learning Reduced expression of MAPK/ERK genes in perinatal arsenic-exposed offspring induced by glucocorticoid receptor deficits.ChIP-Seq analysis of the adult male mouse brain after developmental exposure to arsenic Perinatal exposure to 50 ppb sodium arsenate induces hypothalamic-pituitary-adrenal axis dysregulation in male C57BL/6 mice Prenatal arsenic exposure alters the programming of the glucocorticoid signaling system during embryonic development.Sex-Dependent effects of developmental arsenic exposure on methylation capacity and methylation regulation of the glucocorticoid receptor system in the embryonic mouse brain.Arsenite activates NFκB through induction of C-reactive protein.Prenatal Exposure to Arsenic Impairs Behavioral Flexibility and Cortical Structure in Mice.Child Intelligence and Reductions in Water Arsenic and Manganese: A Two-Year Follow-up Study in Bangladesh.Modeling neurodevelopmental cognitive deficits in tasks with cross-species translational validity Cortical Astrocytes Acutely Exposed to the Monomethylarsonous Acid (MMAIII) Show Increased Pro-inflammatory Cytokines Gene Expression that is Consistent with APP and BACE-1: Over-expression.Evidence of hormesis on human neuronal SK-N-BE cells treated with sodium arsenate: impact at the mitochondrial level.The Effect of Chronic Arsenic Exposure in Zebrafish.The Effects of Arsenic Exposure on Neurological and Cognitive Dysfunction in Human and Rodent Studies: A Review Comparison of neurobehavioral and biochemical effects in rats exposed to dusts from copper smelter plant at different locations.Assessment of neurobehavioral and biochemical effects in rats exposed to copper smelter dusts.Endocrine active metals, prenatal stress and enhanced neurobehavioral disruption.Effects of developmental arsenite exposure on hippocampal synapses in mouse offspring.Arsenic exposure during embryonic development alters the expression of the long noncoding RNA growth arrest specific-5 (Gas5) in a sex-dependent manner.Arsenic induces members of the mmu-miR-466-669 cluster which reduces NeuroD1 expression.Tissue-specific and dose-related accumulation of arsenic in mouse offspring following maternal consumption of arsenic-contaminated water.

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Learning deficits in C57BL/6J mice following perinatal arsenic exposure: consequence of lower corticosterone receptor levels?

http://www.wikidata.org/entity/Q37392258

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 13 September 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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J.PBB.2009.09.006

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Pharmacology, Biochemistry and Behavior

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Learning deficits in C57BL/6J ...... orticosterone receptor levels?

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Abdul-Mehdi S Ali

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Andrea M Allan

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Ebany J Martinez-Finley

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P2860

Arsenic alters the function of the glucocorticoid receptor as a transcription factor

Maternal stress modulates the effects of developmental lead exposure

Water arsenic exposure and children's intellectual function in Araihazar, Bangladesh

Exposure to arsenic and lead and neuropsychological development in Mexican children

Corticosteroids and cognition

Physiology and neurobiology of stress and adaptation: central role of the brain

Molecular basis for effects of carcinogenic heavy metals on inducible gene expression

Acute activation of hippocampal glucocorticoid receptors results in different waves of gene expression throughout time

Arsenic at very low concentrations alters glucocorticoid receptor (GR)-mediated gene activation but not GR-mediated gene repression: complex dose-response effects are closely correlated with levels of activated GR and require a functional GR DNA bin

Fear conditioning is associated with altered integration of PLC and ERK signaling in the hippocampus

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271-277

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scholarly article

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10.1016/J.PBB.2009.09.006

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