Activating mutations of the c-Ha-ras protooncogene in chemically induced hepatomas of the male B6C3 F1 mouse.
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Mechanisms of multistep carcinogenesis and carcinogen risk assessmentActivated neu oncogene sequences in primary tumors of the peripheral nervous system induced in rats by transplacental exposure to ethylnitrosoureaMutation of beta-catenin is an early event in chemically induced mouse hepatocellular carcinogenesisData requirements for risk assessment of furan in food.Influence of nucleotide excision repair on N-hydroxy-2-acetylaminofluorene-induced mutagenesis studied in lambda lacZ-transgenic mice.Differential gene expression during multistage carcinogenesis.Activation of ras oncogene in aflatoxin-induced rat liver carcinogenesis.Metabolic activation capacity of neonatal mice in relation to the neonatal mouse tumorigenicity bioassay.Paradoxical effects of phenobarbital on mouse hepatocarcinogenesis.Activation of cellular oncogenes by chemical carcinogens in Syrian hamster embryo fibroblasts.Molecular analysis of O6-substituted guanine-induced mutagenesis of ras oncogenesA potent modifier of liver cancer risk on distal mouse chromosome 1: linkage analysis and characterization of congenic lines.Possible etiologic mechanisms in chemical carcinogenesisCellular and molecular mechanisms of multistep carcinogenesis: relevance to carcinogen risk assessmentCarcinogenesis studies in rodents for evaluating risks associated with chemical carcinogens in aquatic food animalsThe role of non-ras transforming genes in chemical carcinogenesisFrequency and spectrum of c-Ki-ras mutations in human sporadic colon carcinoma, carcinomas arising in ulcerative colitis, and pancreatic adenocarcinoma.Minimal cooperation between mutant Hras and c-myc or TGFα in the regulation of mouse hepatocyte growth or transformation in vivo.Mutant Hras(G12V) and Kras(G12D) have overlapping, but non-identical effects on hepatocyte growth and transformation frequency in transgenic miceHepatitis B virus infection and primary hepatocellular carcinoma.Conditional transformation of mouse liver epithelial cells. An in vitro model for analysis of genetic events in hepatocarcinogenesisVinyl chloride-a classical industrial toxicant of new interest.The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis.AlkB homologue 2-mediated repair of ethenoadenine lesions in mammalian DNA.Tumorigenicity of acrylamide and its metabolite glycidamide in the neonatal mouse bioassay.Transforming c-Ki-ras mutation is a preneoplastic event in mouse mammary carcinogenesis induced in vitro by N-methyl-N-nitrosoureaH-ras activation in benign and self-regressing skin tumors (keratoacanthomas) in both humans and an animal model systemLack of ecotropic virus involvement in induction of lymphomas in DBA/2J mice by 7,12-dimethylbenz(a)anthracene.Confocal laser scanning microscopy of oncogene localization in rainbow trout cell lines derived from normal and tumor tissue.Tumorigenicity of simian virus 40-hepatocyte cell lines: effect of in vitro and in vivo passage on expression of liver-specific genes and oncogenes.Multiple genes exhibit phenobarbital-induced constitutive active/androstane receptor-mediated DNA methylation changes during liver tumorigenesis and in liver tumors.Roles of UDP-glucuronosyltransferases in chemical carcinogenesis.Mutational activation of the c-Ha-ras gene in liver tumors of different rodent strains: correlation with susceptibility to hepatocarcinogenesisActivated protooncogenes in human lung tumors from smokers.Multilineage somatic activating mutations in HRAS and NRAS cause mosaic cutaneous and skeletal lesions, elevated FGF23 and hypophosphatemia.Identification of an activated c-Ki-ras oncogene in rat liver tumors induced by aflatoxin B1.Pathobiology of chemical hepatocarcinogenesis: recent progress and perspectives. Part II. Metabolic and molecular changes.Carcinogen-induced mutations in the mouse c-Ha-ras gene provide evidence of multiple pathways for tumor progression.Evaluation of in vivo mutagenesis for assessing the health risk of air pollutants.Human carcinogens so far identified.
P2860
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P2860
Activating mutations of the c-Ha-ras protooncogene in chemically induced hepatomas of the male B6C3 F1 mouse.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1986
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@en
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@nl
type
label
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@en
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@nl
prefLabel
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@en
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@nl
P2093
P2860
P356
P1476
Activating mutations of the c- ...... mas of the male B6C3 F1 mouse.
@en
P2093
E C Miller
J A Miller
M W Anderson
R W Wiseman
S J Stowers
P2860
P304
P356
10.1073/PNAS.83.16.5825
P407
P577
1986-08-01T00:00:00Z