Infection of neurons and encephalitis after intracranial inoculation of herpes simplex virus requires the entry receptor nectin-1 - Wikidata - awgldk - TriplyDB

Infection of neurons and encephalitis after intracranial inoculation of herpes simplex virus requires the entry receptor nectin-1

Infection of neurons and encephalitis after intracranial inoculation of herpes simplex virus requires the entry receptor nectin-1

http://www.wikidata.org/entity/Q37394353

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Zika Fetal Neuropathogenesis: Etiology of a Viral Syndrome Listeria monocytogenes internalin B activates junctional endocytosis to accelerate intestinal invasion A key role for nectin-1 in the ventral hippocampus in contextual fear memory.Glycoprotein D actively induces rapid internalization of two nectin-1 isoforms during herpes simplex virus entry Distribution of cellular HSV-1 receptor expression in human brain.Viral encephalomyelitis.Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells.A single-amino-acid substitution in herpes simplex virus 1 envelope glycoprotein B at a site required for binding to the paired immunoglobulin-like type 2 receptor alpha (PILRalpha) abrogates PILRalpha-dependent viral entry and reduces pathogenesis Herpes virus fusion and entry: a story with many characters.Herpes simplex virus serotype and entry receptor availability alter CNS disease in a mouse model of neonatal HSV Crystal structure of herpes simplex virus 2 gD bound to nectin-1 reveals a conserved mode of receptor recognition.Herpesvirus entry mediator and nectin-1 mediate herpes simplex virus 1 infection of the murine cornea.Fusing structure and function: a structural view of the herpesvirus entry machinery.The number of alphaherpesvirus particles infecting axons and the axonal protein repertoire determines the outcome of neuronal infection Herpes B virus utilizes human nectin-1 but not HVEM or PILRα for cell-cell fusion and virus entry.Role of Nectin-1 and Herpesvirus Entry Mediator as Cellular Receptors for Herpes Simplex Virus 1 on Primary Murine Dermal Fibroblasts.The signaling networks of the herpesvirus entry mediator (TNFRSF14) in immune regulation Herpesvirus entry mediator on radiation-resistant cell lineages promotes ocular herpes simplex virus 1 pathogenesis in an entry-independent manner.A herpes simplex virus 2 glycoprotein D mutant generated by bacterial artificial chromosome mutagenesis is severely impaired for infecting neuronal cells and infects only Vero cells expressing exogenous HVEM.Herpesvirus entry mediator is a serotype specific determinant of pathogenesis in ocular herpes.Pathogenesis of neonatal herpes simplex 2 disease in a mouse model is dependent on entry receptor expression and route of inoculation.Directional spread of alphaherpesviruses in the nervous system.The Type I Interferon Response Determines Differences in Choroid Plexus Susceptibility between Newborns and Adults in Herpes Simplex Virus Encephalitis.Herpes simplex virus 1 targets the murine olfactory neuroepithelium for host entry Herpesvirus transport to the nervous system and back again.Participation of 3-O-sulfated heparan sulfates in the protection of macrophages by herpes simplex virus-1 glycoprotein D and cyclophilin B against apoptosis.Induction of conformational changes at the N-terminus of herpes simplex virus glycoprotein D upon binding to HVEM and nectin-1 Herpes simplex virus infects most cell types in vitro: clues to its success Animal models of herpes simplex virus immunity and pathogenesis.Herpes Simplex Virus 1 Enters Human Keratinocytes by a Nectin-1-Dependent, Rapid Plasma Membrane Fusion Pathway That Functions at Low Temperature.Herpesvirus Entry Mediator and Ocular Herpesvirus Infection: More than Meets the Eye.Herpes simplex virus glycoprotein D relocates nectin-1 from intercellular contacts.Initial Contact: The First Steps in Herpesvirus Entry.Innate Immune Mechanisms and Herpes Simplex Virus Infection and Disease.Monoclonal Antibodies, Derived from Humans Vaccinated with the RV144 HIV Vaccine Containing the HVEM Binding Domain of Herpes Simplex Virus (HSV) Glycoprotein D, Neutralize HSV Infection, Mediate Antibody-Dependent Cellular Cytotoxicity, and Protect Unique glycan signatures regulate adeno-associated virus tropism in the developing brain.Formation of fibroblastic reticular network in the brain after infection with neurovirulent murine coronavirus.Implication of Soluble Forms of Cell Adhesion Molecules in Infectious Disease and Tumor: Insights from Transgenic Animal Models.Insights into the pathogenesis of herpes simplex encephalitis from mouse models

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Infection of neurons and encephalitis after intracranial inoculation of herpes simplex virus requires the entry receptor nectin-1

http://www.wikidata.org/entity/Q37394353

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 05 October 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Infection of neurons and encep ...... es the entry receptor nectin-1

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Infection of neurons and encep ...... s the entry receptor nectin-1.

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type

label

Infection of neurons and encep ...... es the entry receptor nectin-1

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Infection of neurons and encep ...... s the entry receptor nectin-1.

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prefLabel

Infection of neurons and encep ...... es the entry receptor nectin-1

@en

Infection of neurons and encep ...... s the entry receptor nectin-1.

@nl

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PNAS.0908892106

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Proceedings of the National Academy of Sciences of the United States of America

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Infection of neurons and encep ...... es the entry receptor nectin-1

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Ghazal Banisadr

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Kelly Glajch

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Patricia G Spear

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Pavel Osten

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Richard J Miller

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Sarah J Kopp

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Ulrike E Maurer

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P2860

A cell surface protein with herpesvirus entry activity (HveB) confers susceptibility to infection by mutants of herpes simplex virus type 1, herpes simplex virus type 2, and pseudorabies virus

PILRalpha is a herpes simplex virus-1 entry coreceptor that associates with glycoprotein B

The murine homolog (Mph) of human herpesvirus entry protein B (HveB) mediates entry of pseudorabies virus but not herpes simplex virus types 1 and 2

The principal neuronal gD-type 3-O-sulfotransferases and their products in central and peripheral nervous system tissues

Porcine HveC, a member of the highly conserved HveC/nectin 1 family, is a functional alphaherpesvirus receptor

A novel role for 3-O-sulfated heparan sulfate in herpes simplex virus 1 entry

Comparative usage of herpesvirus entry mediator A and nectin-1 by laboratory strains and clinical isolates of herpes simplex virus

Entry of herpes simplex virus type 1 into primary sensory neurons in vitro is mediated by Nectin-1/HveC

Roles of cell-adhesion molecules nectin 1 and nectin 3 in ciliary body development

Native 3D intermediates of membrane fusion in herpes simplex virus 1 entry.

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17916-17920

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scholarly article

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10.1073/PNAS.0908892106

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42

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106

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2009-10-05T00:00:00Z

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26871217

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19805039

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2764878

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