Acute lung injury edema fluid decreases net fluid transport across human alveolar epithelial type II cells - Wikidata - awgldk - TriplyDB

Acute lung injury edema fluid decreases net fluid transport across human alveolar epithelial type II cells

Acute lung injury edema fluid decreases net fluid transport across human alveolar epithelial type II cells

http://www.wikidata.org/entity/Q37394874

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Randomized, placebo-controlled clinical trial of an aerosolized β₂-agonist for treatment of acute lung injury.Proteomic and computational analysis of bronchoalveolar proteins during the course of the acute respiratory distress syndrome Inflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections.Cultured alveolar epithelial cells from septic rats mimic in vivo septic lung Conditioned media from mesenchymal stromal cells restore sodium transport and preserve epithelial permeability in an in vitro model of acute alveolar injury.miR-206 inhibits FN1 expression and proliferation and promotes apoptosis of rat type II alveolar epithelial cells.Aging mesenchymal stem cells fail to protect because of impaired migration and antiinflammatory response.Resolution of pulmonary edema. Thirty years of progress.Regulated gene expression in cultured type II cells of adult human lung Allogeneic human mesenchymal stem cells restore epithelial protein permeability in cultured human alveolar type II cells by secretion of angiopoietin-1.Proteolytic release of the receptor for advanced glycation end products from in vitro and in situ alveolar epithelial cells.Viral replication and innate host responses in primary human alveolar epithelial cells and alveolar macrophages infected with influenza H5N1 and H1N1 viruses Acute lung injury: epidemiology, pathogenesis, and treatment.The proteome of Hypobaric Induced Hypoxic Lung: Insights from Temporal Proteomic Profiling for Biomarker Discovery Resolvins Decrease Oxidative Stress Mediated Macrophage and Epithelial Cell Interaction through Decreased Cytokine Secretion.Unexpected Role for Adaptive αβTh17 Cells in Acute Respiratory Distress Syndrome.Concise review: Mesenchymal stem cells for acute lung injury: role of paracrine soluble factors.IL-8 inhibits cAMP-stimulated alveolar epithelial fluid transport via a GRK2/PI3K-dependent mechanism.Human mesenchymal stromal cells reduce influenza A H5N1-associated acute lung injury in vitro and in vivo.Mesenchymal stem cells ameliorate inflammatory cytokine-induced impairment of AT-II cells through a keratinocyte growth factor-dependent PI3K/Akt/mTOR signaling pathway Autologous transplantation of adipose-derived stromal cells ameliorates ventilator-induced lung injury in rats The Fas/FasL pathway impairs the alveolar fluid clearance in mouse lungs.Allogeneic human mesenchymal stem cells for treatment of E. coli endotoxin-induced acute lung injury in the ex vivo perfused human lung.Potential application of mesenchymal stem cells in acute lung injury.Alveolar Fluid Clearance in Pathologically Relevant Conditions: In Vitro and In Vivo Models of Acute Respiratory Distress Syndrome.Ion channels in inflammation.Role of CCR2 and IL-8 in acute lung injury: a new mechanism and therapeutic target.Mesenchymal stem cells and acute lung injury.Paracrine factors from mesenchymal stem cells: a proposed therapeutic tool for acute lung injury and acute respiratory distress syndrome.The role of stretch-activated ion channels in acute respiratory distress syndrome: finally a new target?Cytokine-Regulation of Na+-K+-Cl- Cotransporter 1 and Cystic Fibrosis Transmembrane Conductance Regulator-Potential Role in Pulmonary Inflammation and Edema Formation.Sevoflurane reduces severity of acute lung injury possibly by impairing formation of alveolar oedema.Antibacterial effect of human mesenchymal stem cells is mediated in part from secretion of the antimicrobial peptide LL-37.Reduction in systemic epithelial ion transport in septicemia-related pulmonary edema due to changes in amiloride-insensitive sodium transport?The severity of shock is associated with impaired rates of net alveolar fluid clearance in clinical acute lung injury.Priming with endotoxin increases acute lung injury in mice by enhancing the severity of lung endothelial injury.A FIM study to assess safety and exposure of inhaled single doses of AP301-A specific ENaC channel activator for the treatment of acute lung injury.Report of the ISHLT Working Group on Primary Lung Graft Dysfunction Part III: Mechanisms: A 2016 Consensus Group Statement of the International Society for Heart and Lung Transplantation.Specialized Pro-resolving Mediators Regulate Alveolar Fluid Clearance during Acute Respiratory Distress Syndrome.Cycloheximide and lipopolysaccharide downregulate αENaC mRNA via different mechanisms in alveolar epithelial cells.

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Acute lung injury edema fluid decreases net fluid transport across human alveolar epithelial type II cells

http://www.wikidata.org/entity/Q37394874

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 19 June 2007

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Acute lung injury edema fluid ...... eolar epithelial type II cells

@en

Acute lung injury edema fluid ...... olar epithelial type II cells.

@nl

type

label

Acute lung injury edema fluid ...... eolar epithelial type II cells

@en

Acute lung injury edema fluid ...... olar epithelial type II cells.

@nl

prefLabel

Acute lung injury edema fluid ...... eolar epithelial type II cells

@en

Acute lung injury edema fluid ...... olar epithelial type II cells.

@nl

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Journal of Biological Chemistry

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Acute lung injury edema fluid ...... eolar epithelial type II cells

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P2093

Gregory Dolganov

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Jae W Lee

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Richard D Fremont

http://www.w3.org/2001/XMLSchema#string

Xiaohui Fang

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P2860

Fas and fas ligand are up-regulated in pulmonary edema fluid and lung tissue of patients with acute lung injury and the acute respiratory distress syndrome

Hypoxia and beta 2-agonists regulate cell surface expression of the epithelial sodium channel in native alveolar epithelial cells

Incidence and outcomes of acute lung injury

The acute respiratory distress syndrome

The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination

Expression of highly selective sodium channels in alveolar type II cells is determined by culture conditions.

Sodium channels in alveolar epithelial cells: molecular characterization, biophysical properties, and physiological significance.

An expanded definition of the adult respiratory distress syndrome.

Functional ion channels in pulmonary alveolar type I cells support a role for type I cells in lung ion transport.

Lung epithelial fluid transport and the resolution of pulmonary edema.

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24109-24119

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scholarly article

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10.1074/JBC.M700821200

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33

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282

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Julie A. Bastarache

Lorraine B. Ware

Michael Matthay

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2007-06-19T00:00:00Z

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6257569

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17580309

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2765119

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