Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease. - Wikidata - awgldk - TriplyDB

Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.

Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.

http://www.wikidata.org/entity/Q37396863

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Spatiotemporal Regulation of Hsp90-Ligand Complex Leads to Immune Activation Targeted therapeutics in SLE: emerging strategies to modulate the interferon pathway Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic Cells The regulation of inflammation by interferons and their STATs Role of type I interferons in the activation of autoreactive B cells Cardiovascular disease in lupus: insights and updates Molecular Mechanism for p202-Mediated Specific Inhibition of AIM2 Inflammasome Activation Modelling clinical systemic lupus erythematosus: similarities, differences and success stories A BRISC-SHMT complex deubiquitinates IFNAR1 and regulates interferon responses Type I interferon: friend or foe?TLR ligands up-regulate Trex1 expression in murine conventional dendritic cells through type I Interferon and NF-κB-dependent signaling pathways Type I interferon receptor deficiency prevents murine Sjogren's syndrome Autoimmunity-associated protein tyrosine phosphatase PEP negatively regulates IFN-α receptor signaling Autoantibody-dependent and autoantibody-independent roles for B cells in systemic lupus erythematosus: past, present, and future Nucleic acid sensing receptors in systemic lupus erythematosus: development of novel DNA- and/or RNA-like analogues for treating lupus.An essential role of caspase 1 in the induction of murine lupus and its associated vascular damage Genetic evidence for the role of plasmacytoid dendritic cells in systemic lupus erythematosus.Activation of type I interferon pathway in systemic lupus erythematosus: association with distinct clinical phenotypes.TLR recognition of self nucleic acids hampers glucocorticoid activity in lupus.Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage Requirements for innate immune pathways in environmentally induced autoimmunity.B Cell and BAFF dependence of IFN-α-exaggerated disease in systemic lupus erythematosus-prone NZM 2328 mice.The dysregulation of cytokine networks in systemic lupus erythematosus.Pathogenesis and potential therapeutic targets in systemic lupus erythematosus: from bench to bedside The role of cytokine in the lupus nephritis.Inflammasome activation of IL-18 results in endothelial progenitor cell dysfunction in systemic lupus erythematosus.Cytokine disturbances in systemic lupus erythematosus.New insights into type I interferon and the immunopathogenesis of persistent viral infections.Tolerance to apoptotic cells is regulated by indoleamine 2,3-dioxygenase.Type I interferon-dependent CD86(high) marginal zone precursor B cells are potent T cell costimulators in mice Myeloid dendritic cells from B6.NZM Sle1/Sle2/Sle3 lupus-prone mice express an IFN signature that precedes disease onset Siglec-H protects from virus-triggered severe systemic autoimmunity.A fully human monoclonal antibody with novel binding epitope and excellent neutralizing activity to multiple human IFN-α subtypes: A candidate therapy for systemic lupus erythematosus.Nucleic acid-containing amyloid fibrils potently induce type I interferon and stimulate systemic autoimmunity.A pathogenic IFNα, BLyS and IL-17 axis in Systemic Lupus Erythematosus patients.Wiskott-Aldrich syndrome protein-mediated actin dynamics control type-I interferon production in plasmacytoid dendritic cells.Estrogen Receptor α Deficiency Modulates TLR Ligand-Mediated PDC-TREM Expression in Plasmacytoid Dendritic Cells in Lupus-Prone Mice.Taming lupus-a new understanding of pathogenesis is leading to clinical advances.Exacerbated autoimmunity in the absence of TLR9 in MRL.Fas(lpr) mice depends on Ifnar1 Impact of the Type I Interferon Receptor on the Global Gene Expression Program During the Course of Dendritic Cell Maturation Induced by Polyinosinic Polycytidylic Acid.

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Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.

http://www.wikidata.org/entity/Q37396863

description

article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 07 October 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Deficiency of type I IFN recep ...... ion and protects from disease.

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Deficiency of type I IFN recep ...... ion and protects from disease.

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type

label

Deficiency of type I IFN recep ...... ion and protects from disease.

@en

Deficiency of type I IFN recep ...... ion and protects from disease.

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prefLabel

Deficiency of type I IFN recep ...... ion and protects from disease.

@en

Deficiency of type I IFN recep ...... ion and protects from disease.

@nl

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JIMMUNOL.0803872

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Journal of Immunology

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Deficiency of type I IFN recep ...... ion and protects from disease.

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Alexis Mathian

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Barbara Neas

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Chaim O Jacob

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Chaim Putterman

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Hemant Agrawal

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Michael N Koss

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Noam Jacob

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Sandra Bajana

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Sean Turner

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William Stohl

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P2860

Type I interferon [corrected] gene induction by the interferon regulatory factor family of transcription factors

Interferon-inducible gene expression signature in peripheral blood cells of patients with severe lupus

DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL

Interferon and granulopoiesis signatures in systemic lupus erythematosus blood

NZM2328: a new mouse model of systemic lupus erythematosus with unique genetic susceptibility loci.

Role for nephritogenic T cells in lupus glomerulonephritis: progression to renal failure is accompanied by T cell activation and expansion in regional lymph nodes.

Marked increase in number of dendritic cells in autoimmune-prone (NZW x BXSB)F1 mice with age.

The role of alpha/beta and gamma interferons in development of immunity to influenza A virus in mice

T cell hyperactivity in lupus as a consequence of hyperstimulatory antigen-presenting cells.

Paucity of clinical disease despite serological autoimmunity and kidney pathology in lupus-prone New Zealand mixed 2328 mice deficient in BAFF

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6021-6029

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scholarly article

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10.4049/JIMMUNOL.0803872

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9

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Esther Carreras

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2009-10-07T00:00:00Z

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26879054

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19812195

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