Evasion of alternative complement pathway by Trypanosoma cruzi results from inefficient binding of factor B.
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Evasion and Immuno-Endocrine Regulation in Parasite Infection: Two Sides of the Same Coin in Chagas Disease?Immune Evasion Strategies of Trypanosoma cruziSiglecs and immune regulationMonitoring of the Parasite Load in the Digestive Tract of Rhodnius prolixus by Combined qPCR Analysis and Imaging Techniques Provides New Insights into the Trypanosome Life CycleInfectious diseases associated with complement deficiencies.Inefficient complement system clearance of Trypanosoma cruzi metacyclic trypomastigotes enables resistant strains to invade eukaryotic cellsComplement control protein factor H: the good, the bad, and the inadequate.Human mannose-binding protein activates the alternative complement pathway and enhances serum bactericidal activity on a mannose-rich isolate of Salmonella.Complement component C1q enhances invasion of human mononuclear phagocytes and fibroblasts by Trypanosoma cruzi trypomastigotes.Discovery and verification of osteopontin and Beta-2-microglobulin as promising markers for staging human African trypanosomiasisInhibitory effects of Trypanosoma cruzi sialoglycoproteins on CD4+ T cells are associated with increased susceptibility to infectionChagas' disease.Three-dimensional structures in the design of therapeutics targeting parasitic protozoa: reflections on the past, present and future.Amastigotes of Trypanosoma cruzi escape destruction by the terminal complement components.Developmentally regulated expression by Trypanosoma cruzi of molecules that accelerate the decay of complement C3 convertases.The Role of Sialic Acid-Binding Receptors (Siglecs) in the Immunomodulatory Effects of Trypanosoma cruzi Sialoglycoproteins on the Protective Immunity of the Host.The Complement System: A Prey of Trypanosoma cruzi.Developmentally-regulated virulence factors of Trypanosoma cruzi and their relationship to evasion of host defences.Identification of C3 acceptors responsible for complement activation in Crithidia fasciculata.The emerging role of complement lectin pathway in trypanosomatids: molecular bases in activation, genetic deficiencies, susceptibility to infection, and complement system-based therapeutics.
P2860
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P2860
Evasion of alternative complement pathway by Trypanosoma cruzi results from inefficient binding of factor B.
description
article científic
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article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on September 1986
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Evasion of alternative complem ...... efficient binding of factor B.
@en
Evasion of alternative complem ...... efficient binding of factor B.
@nl
type
label
Evasion of alternative complem ...... efficient binding of factor B.
@en
Evasion of alternative complem ...... efficient binding of factor B.
@nl
prefLabel
Evasion of alternative complem ...... efficient binding of factor B.
@en
Evasion of alternative complem ...... efficient binding of factor B.
@nl
P2093
P2860
P356
P1476
Evasion of alternative complem ...... efficient binding of factor B.
@en
P2093
P2860
P304
P356
10.1073/PNAS.83.17.6593
P407
P577
1986-09-01T00:00:00Z