Aromatase excess in cancers of breast, endometrium and ovary.
about
Adipocytes: impact on tumor growth and potential sites for therapeutic interventionAssessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge aheadCYP19A1 promoter methylation in saliva associated with milestones of pubertal timing in urban girlsMulti-variant pathway association analysis reveals the importance of genetic determinants of estrogen metabolism in breast and endometrial cancer susceptibility.Regulation of aromatase induction by nuclear receptor coregulator PELP1Melatonin modulation of crosstalk among malignant epithelial, endothelial and adipose cells in breast cancer (Review).Estrogen-dependent and estrogen-independent mechanisms contribute to AIB1-mediated tumor formationS2R(Pgrmc1): the cytochrome-related sigma-2 receptor that regulates lipid and drug metabolism and hormone signaling.The transdermal patches for site-specific delivery of letrozole: a new option for breast cancer therapyConcentration of endogenous estrogens and estrogen metabolites in the NCI-60 human tumor cell lines.Weight loss reduces breast ductal fluid estrogens in obese postmenopausal women: a single arm intervention pilot study.Association of two common single-nucleotide polymorphisms in the CYP19A1 locus and ovarian cancer risk.Proteomic signatures of acquired letrozole resistance in breast cancer: suppressed estrogen signaling and increased cell motility and invasiveness.Metformin inhibits aromatase via an extracellular signal-regulated kinase-mediated pathway.Altered expression of ERs, aromatase, and COX2 connected to estrogen action in type 1 endometrial cancer biologyGenetic variation in CYP19A1 and risk of breast cancer and fibrocystic breast conditions among women in Shanghai, China.Sushi Domain-Containing Protein 3: A Potential Target for Breast Cancer.Effects of Neonicotinoids on Promoter-Specific Expression and Activity of Aromatase (CYP19) in Human Adrenocortical Carcinoma (H295R) and Primary Umbilical Vein Endothelial (HUVEC) Cells.Regulation of vascular endothelial growth factor by melatonin in human breast cancer cells.Melatonin interferes in the desmoplastic reaction in breast cancer by regulating cytokine production.Excessive levels of diverse phytoestrogens can modulate steroidogenesis and cell migration of KGN human granulosa-derived tumor cells.Ghrelin and des-acyl ghrelin inhibit aromatase expression and activity in human adipose stromal cells: suppression of cAMP as a possible mechanism.Melatonin promotes differentiation of 3T3-L1 fibroblasts.Lack of genomic rearrangements involving the aromatase gene CYP19A1 in breast cancer.
P2860
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P2860
Aromatase excess in cancers of breast, endometrium and ovary.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 24 May 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Aromatase excess in cancers of breast, endometrium and ovary.
@en
Aromatase excess in cancers of breast, endometrium and ovary.
@nl
type
label
Aromatase excess in cancers of breast, endometrium and ovary.
@en
Aromatase excess in cancers of breast, endometrium and ovary.
@nl
prefLabel
Aromatase excess in cancers of breast, endometrium and ovary.
@en
Aromatase excess in cancers of breast, endometrium and ovary.
@nl
P2093
P2860
P1476
Aromatase excess in cancers of breast, endometrium and ovary.
@en
P2093
Bertan Yilmaz
Bilgin Gurates
Erica Marsh
Erkut Attar
Gonca Imir
Hiroki Utsunomiya
Hiroshi Ishikawa
P2860
P356
10.1016/J.JSBMB.2007.05.027
P577
2007-05-24T00:00:00Z