Restoration of skeletal muscle ischemia-reperfusion injury in humanized immunodeficient mice. - Wikidata - awgldk - TriplyDB

Restoration of skeletal muscle ischemia-reperfusion injury in humanized immunodeficient mice.

Restoration of skeletal muscle ischemia-reperfusion injury in humanized immunodeficient mice.

http://www.wikidata.org/entity/Q37399232

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Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17 Ischemia/Reperfusion.Prevention of intestinal ischemia-reperfusion injury in humanized mice.

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Restoration of skeletal muscle ischemia-reperfusion injury in humanized immunodeficient mice.

http://www.wikidata.org/entity/Q37399232

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on August 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Restoration of skeletal muscle ...... umanized immunodeficient mice.

@en

Restoration of skeletal muscle ...... umanized immunodeficient mice.

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type

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Restoration of skeletal muscle ...... umanized immunodeficient mice.

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Restoration of skeletal muscle ...... umanized immunodeficient mice.

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prefLabel

Restoration of skeletal muscle ...... umanized immunodeficient mice.

@en

Restoration of skeletal muscle ...... umanized immunodeficient mice.

@nl

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P356

J.SURG.2009.06.010

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Restoration of skeletal muscle ...... umanized immunodeficient mice.

@en

P2093

Cyrus Ahmadi-Yazdi

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Eric G Sheu

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Francis D Moore

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Jalil Afnan

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Michael C Carroll

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Sean M Oakes

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P2860

Origins and functions of B-1 cells with notes on the role of CD5

CXCL13 is required for B1 cell homing, natural antibody production, and body cavity immunity

Attenuation of skeletal muscle reperfusion injury with intravenous 12 amino acid peptides that bind to pathogenic IgM.

Humanized mice in translational biomedical research.

A new Hu-PBL model for the study of human islet alloreactivity based on NOD-scid mice bearing a targeted mutation in the IL-2 receptor gamma chain gene.

Ischaemia-reperfusion is an event triggered by immune complexes and complement.

Innate response to self-antigen significantly exacerbates burn wound depth.

Innate autoimmunity.

Identification of the target self-antigens in reperfusion injury.

Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

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340-346

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scholarly article

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10.1016/J.SURG.2009.06.010

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146

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2009-08-01T00:00:00Z

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26691387

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19628094

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reperfusion injury

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2767199

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