A Drosophila mutation that eliminates a calcium-dependent potassium current. - Wikidata - awgldk - TriplyDB

A Drosophila mutation that eliminates a calcium-dependent potassium current.

A Drosophila mutation that eliminates a calcium-dependent potassium current.

http://www.wikidata.org/entity/Q37406521

about

Large conductance voltage- and calcium-dependent K+ channel, a distinct member of voltage-dependent ion channels with seven N-terminal transmembrane segments (S0-S6), an extracellular N terminus, and an intracellular (S9-S10) C terminus Control of daily transcript oscillations in Drosophila by light and the circadian clock.SLO-1-channels of parasitic nematodes reconstitute locomotor behaviour and emodepside sensitivity in Caenorhabditis elegans slo-1 loss of function mutants Response to mechanical stress is mediated by the TRPA channel painless in the Drosophila heart Characterization of the Ca2+-gated and voltage-dependent K+-channel Slo-1 of nematodes and its interaction with emodepside Deletion of the Ca2+-activated potassium (BK) alpha-subunit but not the BKbeta1-subunit leads to progressive hearing loss Lithium-Responsive Seizure-Like Hyperexcitability Is Caused by a Mutation in the Drosophila Voltage-Gated Sodium Channel Gene paralytic.De novo transcriptome assembly for the lobster Homarus americanus and characterization of differential gene expression across nervous system tissues.Analysis of temperature-sensitive mutants reveals new genes involved in the courtship song of Drosophila.Homeotic control in Drosophila; the scabrous gene is an in vivo target of Ultrabithorax proteins.A BK (Slo1) channel journey from molecule to physiology.Synaptic excitation is regulated by the postsynaptic dSK channel at the Drosophila larval NMJ.An improved method for accurate and rapid measurement of flight performance in Drosophila SLOB, a SLOWPOKE channel binding protein, regulates insulin pathway signaling and metabolism in Drosophila In vivo functional role of the Drosophila hyperkinetic beta subunit in gating and inactivation of Shaker K+ channels.Cloning and distribution of Ca2+-activated K+ channels in lobster Panulirus interruptus.dyschronic, a Drosophila homolog of a deaf-blindness gene, regulates circadian output and Slowpoke channels.Expression of Drosophila Shaker potassium channels in mammalian cells infected with recombinant vaccinia virus.BK channels regulate sinoatrial node firing rate and cardiac pacing in vivo Functional upregulation of Ca(2+)-activated K(+) channels in the development of substantia nigra dopamine neurons.Slob, a Slowpoke channel-binding protein, modulates synaptic transmission Analysis of conditional paralytic mutants in Drosophila sarco-endoplasmic reticulum calcium ATPase reveals novel mechanisms for regulating membrane excitability.Temperature-sensitive paralytic mutants are enriched for those causing neurodegeneration in Drosophila.Profile of Barry Ganetzky δ/ω-Plectoxin-Pt1a: an excitatory spider toxin with actions on both Ca(2+) and Na(+) channels.From genes to integrative physiology: ion channel and transporter biology in Caenorhabditis elegans.Impaired clock output by altered connectivity in the circadian network.Role of NKCC in BK channel-mediated net K⁺ secretion in the CCD The Cyclooctadepsipeptide Anthelmintic Emodepside Differentially Modulates Nematode, Insect and Human Calcium-Activated Potassium (SLO) Channel Alpha Subunits.Segmental differences in firing properties and potassium currents in Drosophila larval motoneurons.Ways and means for left shifts in the MaxiK channel.Cell-specific regulation of L-WNK1 by dietary K.Charybdotoxin blocks voltage-gated K+ channels in human and murine T lymphocytes.Cytosolic calcium coordinates mitochondrial energy metabolism with presynaptic activity Regulation of cation transport in the distal nephron by mechanical forces.The fundamentals of flying: simple and inexpensive strategies for employing Drosophila genetics in neuroscience teaching laboratories Effects of social isolation on neuromuscular excitability and aggressive behaviors in Drosophila: altered responses by Hk and gsts1, two mutations implicated in redox regulation An unexpected journey: conceptual evolution of mechanoregulated potassium transport in the distal nephron.Cell-specific fine-tuning of neuronal excitability by differential expression of modulator protein isoforms.Regulation of synaptic transmission by presynaptic CaMKII and BK channels.

P2860

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P2860

A Drosophila mutation that eliminates a calcium-dependent potassium current.

http://www.wikidata.org/entity/Q37406521

description

1986 nî lūn-bûn

@nan

1986年の論文

@ja

1986年学术文章

@wuu

1986年学术文章

@zh-cn

1986年学术文章

@zh-hans

1986年学术文章

@zh-my

1986年学术文章

@zh-sg

1986年學術文章

@yue

1986年學術文章

@zh

1986年學術文章

@zh-hant

name

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@en

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@nl

type

label

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@en

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@nl

prefLabel

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@en

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@nl

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P356

PNAS.83.21.8415

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

A Drosophila mutation that eliminates a calcium-dependent potassium current.

@en

P2093

B Ganetzky

http://www.w3.org/2001/XMLSchema#string

C F Wu

http://www.w3.org/2001/XMLSchema#string

T Elkins

http://www.w3.org/2001/XMLSchema#string

P2860

Genetic modification of potassium channels in Drosophila Shaker mutants

Potassium current suppression by quinidine reveals additional calcium currents in neuroblastoma cells

Ca2+ -activated K+ conductance in internally perfused snail neurons is enhanced by protein phosphorylation.

Abnormal action potentials associated with the Shaker complex locus of Drosophila

Mutant analysis shows that the Ca2+-induced K+ current shuts off one type of excitation in Paramecium.

A Drosophila mutant with a temperature-sensitive block in nerve conduction.

Two distinct Ca-dependent K currents in bullfrog sympathetic ganglion cells.

Two mutations of synaptic transmission in Drosophila.

Apamin, a neurotoxin specific for one class of Ca2+-dependent K+ channels.

Apamin blocks certain neurotransmitter-induced increases in potassium permeability.

P304

8415-8419

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scholarly article

P356

10.1073/PNAS.83.21.8415

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P433

21

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83

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2430288

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386939

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