Long-term administration of valacyclovir reduces the number of Epstein-Barr virus (EBV)-infected B cells but not the number of EBV DNA copies per B cell in healthy volunteers.
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Progress and problems in understanding and managing primary Epstein-Barr virus infectionsApproved Antiviral Drugs over the Past 50 YearsAcyclovir Therapy Reduces the CD4+ T Cell Response against the Immunodominant pp65 Protein from Cytomegalovirus in Immune Competent IndividualsTarget DNA detection and quantitation on a single cell with single base resolution.Effect of high versus low oral doses of valacyclovir on herpes simplex virus-1 DNA shedding into tears of latently infected rabbits.A new model of Epstein-Barr virus infection reveals an important role for early lytic viral protein expression in the development of lymphomas.An Epstein-Barr virus encoded inhibitor of Colony Stimulating Factor-1 signaling is an important determinant for acute and persistent EBV infection.In vivo function of the murid herpesvirus-4 ribonucleotide reductase small subunit.Gammaherpesvirus gene expression and DNA synthesis are facilitated by viral protein kinase and histone variant H2AX.Dynamic association of gammaherpesvirus DNA with core histone during de novo lytic infection of primary cells.Ataxia telangiectasia mutated kinase controls chronic gammaherpesvirus infection.Memory B-cell reconstitution following allogeneic hematopoietic stem cell transplantation is an EBV-associated transformation eventCell-based screening assay for antiviral compounds targeting the ability of herpesvirus posttranscriptional regulatory proteins to stabilize viral mRNAs.Valacyclovir: approved and off-label uses for the treatment of herpes virus infections in immunocompetent and immunocompromised adults.Design and development of antivirals and intervention strategies against human herpesviruses using high-throughput approach.EBV Persistence--Introducing the Virus.De novo infection of B cells during murine gammaherpesvirus 68 latencyLeflunomide/teriflunomide inhibit Epstein-Barr virus (EBV)- induced lymphoproliferative disease and lytic viral replication.CTCF and Rad21 act as host cell restriction factors for Kaposi's sarcoma-associated herpesvirus (KSHV) lytic replication by modulating viral gene transcription.Gamma-herpesvirus colonization of the spleen requires lytic replication in B cells.Epstein-Barr virus exanthem in an adult patient with myelodysplastic syndrome.Continuous DNA replication is required for late gene transcription and maintenance of replication compartments in gammaherpesviruses.Transcriptome Analysis and In Situ Hybridization for FcaGHV1 in Feline LymphomaEffect of valacyclovir on EHV-5 viral kinetics in horses with equine multinodular pulmonary fibrosis
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P2860
Long-term administration of valacyclovir reduces the number of Epstein-Barr virus (EBV)-infected B cells but not the number of EBV DNA copies per B cell in healthy volunteers.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Long-term administration of va ...... B cell in healthy volunteers.
@en
Long-term administration of valacyclovir reduces the number of Epstein-Barr virus
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type
label
Long-term administration of va ...... B cell in healthy volunteers.
@en
Long-term administration of valacyclovir reduces the number of Epstein-Barr virus
@nl
prefLabel
Long-term administration of va ...... B cell in healthy volunteers.
@en
Long-term administration of valacyclovir reduces the number of Epstein-Barr virus
@nl
P2093
P2860
P356
P1433
P1476
Long-term administration of va ...... r B cell in healthy volunteers
@en
P2093
Dean Follmann
Jeffrey I Cohen
Patricia Hohman
Stephen K Tyring
Yo Hoshino
P2860
P304
11857-11861
P356
10.1128/JVI.01005-09
P407
P577
2009-09-09T00:00:00Z