Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
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The dysconnection hypothesis (2016)Catechol-O-methyltransferase and genetic variation under hemizygosity22q11 deletion syndrome: a review of the neuropsychiatric features and their neurobiological basis.Germline recessive mutations in PI4KA are associated with perisylvian polymicrogyria, cerebellar hypoplasia and arthrogryposisTranscriptome Profiling of Peripheral Blood in 22q11.2 Deletion Syndrome Reveals Functional Pathways Related to Psychosis and Autism Spectrum DisorderCOMT Genetic Reduction Produces Sexually Divergent Effects on Cortical Anatomy and Working Memory in Mice and Humans.Oppositional COMT Val158Met effects on resting state functional connectivity in adolescents and adults.Neural correlates of reward processing in adults with 22q11 deletion syndrome.Association study of single nucleotide polymorphism rs165599 of COMT gene, with schizophrenia and bipolar mood disorder in the south-west of Iran.Genotype-Dependent Effects of COMT Inhibition on Cognitive Function in a Highly Specific, Novel Mouse Model of Altered COMT Activity.Genetic moderation of the effects of cannabis: catechol-O-methyltransferase (COMT) affects the impact of Δ9-tetrahydrocannabinol (THC) on working memory performance but not on the occurrence of psychotic experiences.IQ and hemizygosity for the Val158 Met functional polymorphism of COMT in 22q11DS.Adolescence is the starting point of sex-dichotomous COMT genetic effects.Association of oxidative stress-related genes with idiopathic recurrent miscarriage.Mother-Child Interaction as a Window to a Unique Social Phenotype in 22q11.2 Deletion Syndrome and in Williams Syndrome.COMT haplotypes, catecholamine metabolites in plasma and clinical response in schizophrenic and bipolar patients.Modulative effects of COMT haplotype on age-related associations with brain morphology.Replication analyses of four chromosomal deletions with schizophrenia via independent large-scale meta-analyses.The Psychosis Spectrum in 22q11.2 Deletion Syndrome Is Comparable to That of Nondeleted Youths.Schizophrenia in DiGeorge Syndrome: A Unique Case Report
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P2860
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
@en
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome.
@nl
type
label
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
@en
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome.
@nl
prefLabel
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
@en
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome.
@nl
P2093
P2860
P50
P1476
Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome
@en
P2093
Amanda J Law
Amos Frisch
Barbara K Lipska
Doron Gothelf
Elena Michaelovsky
Jingshan Chen
Miri Carmel
Omer Zarchi
Renee Ren-Patterson
P2860
P304
P356
10.1016/J.BIOPSYCH.2013.07.021
P407
P577
2013-08-28T00:00:00Z