Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques. - Wikidata - awgldk - TriplyDB

Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques.

Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques.

http://www.wikidata.org/entity/Q37416056

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Monocytes in atherosclerosis: subsets and functions Induction of dendritic cell-like phenotype in macrophages during foam cell formation Integrin alphaDbeta2, an adhesion receptor up-regulated on macrophage foam cells, exhibits multiligand-binding properties Endocytic receptor LRP together with tPA and PAI-1 coordinates Mac-1-dependent macrophage migration.High density lipoprotein and metabolic disease: Potential benefits of restoring its functional properties Macrophage Heterogeneity and Plasticity: Impact of Macrophage Biomarkers on Atherosclerosis Resolution of inflammation: an integrated view Monocytes as regulators of inflammation and HIV-related comorbidities during cART Regression of atherosclerosis: insights from animal and clinical studies Epigenome-guided analysis of the transcriptome of plaque macrophages during atherosclerosis regression reveals activation of the Wnt signaling pathway Quantitative analysis of monocyte subpopulations in murine atherosclerotic plaques by multiphoton microscopy Divergent JAM-C Expression Accelerates Monocyte-Derived Cell Exit from Atherosclerotic Plaques Inflammatory cell recruitment in cardiovascular disease: murine models and potential clinical applications NF-E2-related factor 2 promotes atherosclerosis by effects on plasma lipoproteins and cholesterol transport that overshadow antioxidant protection Regression of atherosclerosis is characterized by broad changes in the plaque macrophage transcriptome Statins promote the regression of atherosclerosis via activation of the CCR7-dependent emigration pathway in macrophages Contribution of vascular cell-derived cytokines to innate and inflammatory pathways in atherogenesis.Vascular cells contribute to atherosclerosis by cytokine- and innate-immunity-related inflammatory mechanisms.Dynamic T cell-APC interactions sustain chronic inflammation in atherosclerosis Adenosine receptor A2A deficiency in leukocytes increases arterial neointima formation in apolipoprotein E-deficient mice Diabetic conditions promote binding of monocytes to vascular smooth muscle cells and their subsequent differentiation Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent.Noninvasive in vivo imaging of monocyte trafficking to atherosclerotic lesions IFN-gamma and STAT1 arrest monocyte migration and modulate RAC/CDC42 pathways Macrophages create an acidic extracellular hydrolytic compartment to digest aggregated lipoproteins.Deletion of ABCA1 and ABCG1 impairs macrophage migration because of increased Rac1 signaling.Oxidized LDL/CD36 interaction induces loss of cell polarity and inhibits macrophage locomotion.HDL induces the expression of the M2 macrophage markers arginase 1 and Fizz-1 in a STAT6-dependent process.Gene expression changes in foam cells and the role of chemokine receptor CCR7 during atherosclerosis regression in ApoE-deficient mice.Human antimicrobial peptide LL-37 is present in atherosclerotic plaques and induces death of vascular smooth muscle cells: a laboratory study.Innate and adaptive immunity in atherosclerosis.Mechanisms that regulate macrophage burden in atherosclerosis.Surface engineering of quantum dots for in vivo vascular imaging.Macrophage death and defective inflammation resolution in atherosclerosis.Kinetic assessment and therapeutic modulation of metabolic and inflammatory profiles in mice on a high-fat and cholesterol diet Inflammation and its resolution as determinants of acute coronary syndromes.CD36, a scavenger receptor implicated in atherosclerosis.HDL promotes rapid atherosclerosis regression in mice and alters inflammatory properties of plaque monocyte-derived cells.Apolipoprotein A-I mimetic 4F alters the function of human monocyte-derived macrophages.Lysophosphatidic acid in atherosclerotic diseases

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P2860

Emigration of monocyte-derived cells from atherosclerotic lesions characterizes regressive, but not progressive, plaques.

http://www.wikidata.org/entity/Q37416056

description

article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on 27 July 2004

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Emigration of monocyte-derived ...... but not progressive, plaques.

@en

Emigration of monocyte-derived ...... but not progressive, plaques.

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type

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Emigration of monocyte-derived ...... but not progressive, plaques.

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Emigration of monocyte-derived ...... but not progressive, plaques.

@nl

prefLabel

Emigration of monocyte-derived ...... but not progressive, plaques.

@en

Emigration of monocyte-derived ...... but not progressive, plaques.

@nl

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PNAS.0403259101

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Emigration of monocyte-derived ...... but not progressive, plaques.

@en

P2093

Eugene Trogan

http://www.w3.org/2001/XMLSchema#string

Gwendalyn J Randolph

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Jaime Llodrá

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Jianhua Liu

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Véronique Angeli

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P2860

Expanding expression of the 5-lipoxygenase pathway within the arterial wall during human atherogenesis

Atherosclerosis. the road ahead

Reduction of isoprostanes and regression of advanced atherosclerosis by apolipoprotein E.

Adenovirus-mediated gene transfer of human platelet-activating factor-acetylhydrolase prevents injury-induced neointima formation and reduces spontaneous atherosclerosis in apolipoprotein E-deficient mice.

Dendritic cells and their involvement in atherosclerosis.

Identification of the G994--> T missense in exon 9 of the plasma platelet-activating factor acetylhydrolase gene as an independent risk factor for coronary artery disease in Japanese men.

Identification of an intracellular receptor for lysophosphatidic acid (LPA): LPA is a transcellular PPARgamma agonist.

Travellers in many guises: the origins and destinations of dendritic cells.

Role of monocytes in atherogenesis.

The role of the monocyte in atherogenesis: II. Migration of foam cells from atherosclerotic lesions.

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10.1073/PNAS.0403259101

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101

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Edward A Fisher

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2004-07-27T00:00:00Z

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8428526

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15280540

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atherosclerosis

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511052

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