Natural indoles, indole-3-carbinol and 3,3'-diindolymethane, inhibit T cell activation by staphylococcal enterotoxin B through epigenetic regulation involving HDAC expression.
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Natural indoles, indole-3-carbinol (I3C) and 3,3'-diindolylmethane (DIM), attenuate staphylococcal enterotoxin B-mediated liver injury by downregulating miR-31 expression and promoting caspase-2-mediated apoptosisRole of miRNA in the regulation of inflammatory genes in staphylococcal enterotoxin B-induced acute inflammatory lung injury and mortality3,3'-Diindolylmethane attenuates LPS-mediated acute liver failure by regulating miRNAs to target IRAK4 and suppress Toll-like receptor signallingDietary Indoles Suppress Delayed-Type Hypersensitivity by Inducing a Switch from Proinflammatory Th17 Cells to Anti-Inflammatory Regulatory T Cells through Regulation of MicroRNA.3,39-Diindolylmethane Ameliorates Staphylococcal Enterotoxin B–Induced Acute Lung Injury through Alterations in the Expression of MicroRNA that Target Apoptosis and Cell-Cycle Arrest in Activated T Cells.Limited but durable changes to cellular gene expression in a model of latent adenovirus infection are reflected in childhood leukemic cell lines.Natural compound-derived epigenetic regulators targeting epigenetic readers, writers and erasers.RNA-seq Analysis of δ9-Tetrahydrocannabinol-treated T Cells Reveals Altered Gene Expression Profiles That Regulate Immune Response and Cell Proliferation.Induction of specific T helper-9 cells to inhibit glioma cell growth.In vitro analysis of factors influencing CYP1A2 expression as potential determinants of interindividual variation.Inhibition of squamous cancer growth in a mouse model by Staphylococcal enterotoxin B-triggered Th9 cell expansion.Histone and Non-Histone Targets of Dietary Deacetylase Inhibitors.The bounty of nature for changing the cancer landscape.Dietary phytochemicals as epigenetic modifiers in cancer: Promise and challenges.Epigenetics of breast cancer: Modifying role of environmental and bioactive food compounds.Regulation of Trek1 expression in nasal mucosa with allergic rhinitis by specific immunotherapy.Trek1 contributes to maintaining nasal epithelial barrier integrity.Current results on the biological and pharmacological activities of Indole-3-carbinol.Cannabidiol Attenuates Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis Through Induction of Myeloid-Derived Suppressor CellsThe Emerging Spectrum of Early Life Exposure-Related Inflammation and Epigenetic Therapy
P2860
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P2860
Natural indoles, indole-3-carbinol and 3,3'-diindolymethane, inhibit T cell activation by staphylococcal enterotoxin B through epigenetic regulation involving HDAC expression.
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2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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name
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@en
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@nl
type
label
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@en
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@nl
prefLabel
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@en
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@nl
P2093
P2860
P1476
Natural indoles, indole-3-carb ...... ion involving HDAC expression.
@en
P2093
Mitzi Nagarkatti
Philip B Busbee
Prakash S Nagarkatti
P2860
P356
10.1016/J.TAAP.2013.10.022
P577
2013-11-05T00:00:00Z