PI3K, Rho, and ROCK play a key role in hypoxia-induced ATP release and ATP-stimulated angiogenic responses in pulmonary artery vasa vasorum endothelial cells
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The adventitia: essential regulator of vascular wall structure and functionExpression of pannexin isoforms in the systemic murine arterial network.P2Y1 and P2Y13 purinergic receptors mediate Ca2+ signaling and proliferative responses in pulmonary artery vasa vasorum endothelial cellsComplementary effects of extracellular nucleotides and platelet-derived extracts on angiogenesis of vasa vasorum endothelial cells in vitro and subcutaneous Matrigel plugs in vivo.Adenosine A1 receptors promote vasa vasorum endothelial cell barrier integrity via Gi and Akt-dependent actin cytoskeleton remodelingATP release mechanisms of endothelial cell-mediated stimulus-dependent hyperalgesiaWhat is the efficiency of ATP signaling from erythrocytes to regulate distribution of O(2) supply within the microvasculature?Adaptive and maladaptive cardiorespiratory responses to continuous and intermittent hypoxia mediated by hypoxia-inducible factors 1 and 2.Chronic hypoxia impairs extracellular nucleotide metabolism and barrier function in pulmonary artery vasa vasorum endothelial cells.Glycolysis and oxidative phosphorylation are essential for purinergic receptor-mediated angiogenic responses in vasa vasorum endothelial cells.High proliferative potential endothelial colony-forming cells contribute to hypoxia-induced pulmonary artery vasa vasorum neovascularizationNucleotide receptors as targets in the pharmacological enhancement of dermal wound healing.Metabolic sensors and their interplay with cell signalling and transcription.New insights regarding the regulation of chemotaxis by nucleotides, adenosine, and their receptors.Microenvironmental Interaction Between Hypoxia and Endothelial Cells Controls the Migration Ability of Placenta-Derived Mesenchymal Stem Cells via α4 Integrin and Rho Signaling.RhoA-mediated signaling in mechanotransduction of osteoblasts.Adenosine A2A receptor-dependent proliferation of pulmonary endothelial cells is mediated through calcium mobilization, PI3-kinase and ERK1/2 pathways.Hypoxia is an effective stimulus for vesicular release of ATP from human umbilical vein endothelial cells.Adenovirus-mediated RhoA shRNA suppresses growth of esophageal squamous cell carcinoma cells in vitro and in vivo.Hypoxia induces pulmonary arterial fibroblast proliferation, migration, differentiation and vascular remodeling via the PI3K/Akt/p70S6K signaling pathway.
P2860
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P2860
PI3K, Rho, and ROCK play a key role in hypoxia-induced ATP release and ATP-stimulated angiogenic responses in pulmonary artery vasa vasorum endothelial cells
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 August 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
PI3K, Rho, and ROCK play a key ...... vasa vasorum endothelial cells
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PI3K, Rho, and ROCK play a key ...... asa vasorum endothelial cells.
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type
label
PI3K, Rho, and ROCK play a key ...... vasa vasorum endothelial cells
@en
PI3K, Rho, and ROCK play a key ...... asa vasorum endothelial cells.
@nl
prefLabel
PI3K, Rho, and ROCK play a key ...... vasa vasorum endothelial cells
@en
PI3K, Rho, and ROCK play a key ...... asa vasorum endothelial cells.
@nl
P2093
P2860
P1476
PI3K, Rho, and ROCK play a key ...... vasa vasorum endothelial cells
@en
P2093
Adil Anwar
Evgenia V Gerasimovskaya
Heather N Woodward
Kurt R Stenmark
Laimute Taraseviciene-Stewart
Miguel Fragoso
Suzette Riddle
P2860
P304
P356
10.1152/AJPLUNG.00038.2009
P577
2009-08-14T00:00:00Z