Ischemia-induced brain damage is enhanced in human renin and angiotensinogen double-transgenic mice.
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Angiotensin-converting enzyme 2 priming enhances the function of endothelial progenitor cells and their therapeutic efficacy.Histamine H4-receptors inhibit mast cell renin release in ischemia/reperfusion via protein kinase C ε-dependent aldehyde dehydrogenase type-2 activationActivation of the ACE2/Ang-(1-7)/Mas pathway reduces oxygen-glucose deprivation-induced tissue swelling, ROS production, and cell death in mouse brain with angiotensin II overproduction.Bone marrow mononuclear cells exert long-term neuroprotection in a rat model of ischemic stroke by promoting arteriogenesis and angiogenesis.Transfusion of CXCR4-primed endothelial progenitor cells reduces cerebral ischemic damage and promotes repair in db/db diabetic miceElectroacupuncture improves cerebral blood flow and attenuates moderate ischemic injury via Angiotensin II its receptors-mediated mechanism in rats.Mechanisms underlying the cerebral microvascular responses to angiotensin II-induced hypertension.E-NTPDase1/CD39 modulates renin release from heart mast cells during ischemia/reperfusion: a novel cardioprotective role.Protecting against vascular disease in brainCirculating endothelial progenitor cells and cellular membrane microparticles in db/db diabetic mouse: possible implications in cerebral ischemic damageRole of blood cell-associated angiotensin II type 1 receptors in the cerebral microvascular response to ischemic stroke during angiotensin-induced hypertensionNeuroprotective Effect of Scutellarin on Ischemic Cerebral Injury by Down-Regulating the Expression of Angiotensin-Converting Enzyme and AT1 ReceptorAngiotensin converting enzyme 2/Ang-(1-7)/mas axis protects brain from ischemic injury with a tendency of age-dependenceMicrovascular endothelial cells-derived microvesicles imply in ischemic stroke by modulating astrocyte and blood brain barrier function and cerebral blood flow.Effects of endothelial progenitor cell-derived microvesicles on hypoxia/reoxygenation-induced endothelial dysfunction and apoptosis.Neuronal over-expression of ACE2 protects brain from ischemia-induced damageRenin-Angiotensin system hyperactivation can induce inflammation and retinal neural dysfunction.The neuroprotective effect of losartan through inhibiting AT1/ASK1/MKK4/JNK3 pathway following cerebral I/R in rat hippocampal CA1 region.
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P2860
Ischemia-induced brain damage is enhanced in human renin and angiotensinogen double-transgenic mice.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 16 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@en
Ischemia-induced brain damage ...... inogen double-transgenic mice.
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type
label
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@en
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@nl
prefLabel
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@en
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@nl
P2093
P2860
P1476
Ischemia-induced brain damage ...... inogen double-transgenic mice.
@en
P2093
Curt D Sigmund
Guangze Li
James E Olson
Jinju Wang
Shuzhen Chen
Wenfeng Zhang
Yanfang Chen
P2860
P304
P356
10.1152/AJPREGU.91040.2008
P577
2009-09-16T00:00:00Z