Allergic sensitization through the airway primes Th17-dependent neutrophilia and airway hyperresponsiveness. - Wikidata - awgldk - TriplyDB

Allergic sensitization through the airway primes Th17-dependent neutrophilia and airway hyperresponsiveness.

Allergic sensitization through the airway primes Th17-dependent neutrophilia and airway hyperresponsiveness.

http://www.wikidata.org/entity/Q37423124

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Interplay of T Helper 17 Cells with CD4(+)CD25(high) FOXP3(+) Tregs in Regulation of Allergic Asthma in Pediatric Patients Update in asthma 2009 Neuroimmune semaphorin 4A downregulates the severity of allergic response Herbal Medicines for Asthmatic Inflammation: From Basic Researches to Clinical Applications Microbial Ligand Costimulation Drives Neutrophilic Steroid-Refractory Asthma Mechanisms underlying helper T-cell plasticity: implications for immune-mediated disease.Therapeutic interventions in severe asthma The IL-17 pathway as a major therapeutic target in autoimmune diseases NO2 inhalation induces maturation of pulmonary CD11c+ cells that promote antigenspecific CD4+ T cell polarization House Dust Mite Allergen Regulates Constitutive Apoptosis of Normal and Asthmatic Neutrophils via Toll-Like Receptor 4 Neutralization of interleukin-17 suppresses allergic rhinitis symptoms by downregulating Th2 and Th17 responses and upregulating the Treg response.Intraepithelial neutrophils in pediatric severe asthma are associated with better lung function.Schistosoma japonicum infection downregulates house dust mite-induced allergic airway inflammation in mice.Concomitant exposure to ovalbumin and endotoxin augments airway inflammation but not airway hyperresponsiveness in a murine model of asthma.Reducing LPS content in cockroach allergens increases pulmonary cytokine production without increasing inflammation: a randomized laboratory study.Particulate matter-induced airway hyperresponsiveness is lymphocyte dependent Ozone activates pulmonary dendritic cells and promotes allergic sensitization through a Toll-like receptor 4-dependent mechanism Update in environmental and occupational lung diseases 2013 Precision-cut Mouse Lung Slices to Visualize Live Pulmonary Dendritic Cells.Aligning mouse models of asthma to human endotypes of disease.Interleukin-17 regulation: an attractive therapeutic approach for asthma Importance of cytokines in murine allergic airway disease and human asthma.Role of Tyk-2 in Th9 and Th17 cells in allergic asthma.The complex relationship between inflammation and lung function in severe asthma Th17 cells demonstrate stable cytokine production in a proallergic environment Expression of the T helper 17-associated cytokines IL-17A and IL-17F in asthma and COPD.Animal models of allergic airways disease: where are we and where to next?T lymphocyte antigen 4-modified dendritic cell therapy for asthmatic mice guided by the CCR7 chemokine receptor Cbl-b regulates airway mucosal tolerance to aeroallergen.IL-17 in severe asthma. Where do we stand?IL-17A modulates oxidant stress-induced airway hyperresponsiveness but not emphysema.IL-35 production by inducible costimulator (ICOS)-positive regulatory T cells reverses established IL-17-dependent allergic airways disease Echinococcus granulosus infection reduces airway inflammation of mice likely through enhancing IL-10 and down-regulation of IL-5 and IL-17A Trif-dependent induction of Th17 immunity by lung dendritic cells Early immunological response to German cockroach frass exposure induces a Th2/Th17 environment.Cytokine/anti-cytokine therapy - novel treatments for asthma?Airway epithelial NF-κB activation promotes allergic sensitization to an innocuous inhaled antigen The endogenous Th17 response in NO2-promoted allergic airway disease is dispensable for airway hyperresponsiveness and distinct from Th17 adoptive transfer.Inflammasome Activity in Non-Microbial Lung Inflammation.Serum amyloid A activates the NLRP3 inflammasome and promotes Th17 allergic asthma in mice.

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Allergic sensitization through the airway primes Th17-dependent neutrophilia and airway hyperresponsiveness.

http://www.wikidata.org/entity/Q37423124

description

article científic

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bilimsel makale

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scientific article published on 06 August 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Allergic sensitization through ...... nd airway hyperresponsiveness.

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Allergic sensitization through ...... nd airway hyperresponsiveness.

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Allergic sensitization through ...... nd airway hyperresponsiveness.

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Allergic sensitization through ...... nd airway hyperresponsiveness.

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Allergic sensitization through ...... nd airway hyperresponsiveness.

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RCCM.200904-0573OC

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American Journal of Respiratory and Critical Care Medicine

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Allergic sensitization through ...... nd airway hyperresponsiveness.

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Donald N Cook

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Gregory S Whitehead

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Hideki Nakano

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Jay K Kolls

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Meghan E Free

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Rhonda H Wilson

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P2860

The biological functions of T helper 17 cell effector cytokines in inflammation

Dissociation by steroids of eosinophilic inflammation from airway hyperresponsiveness in murine airways

Cutting edge: interleukin 17 signals through a heteromeric receptor complex

IL-17A-producing neutrophil-regulatory Tn lymphocytes

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

MyD88-dependent induction of allergic Th2 responses to intranasal antigen.

Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defense.

IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines.

Neutrophil recruitment by human IL-17 via C-X-C chemokine release in the airways.

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10.1164/RCCM.200904-0573OC

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8

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