N-hydroxybenzimidazole inhibitors of the transcription factor LcrF in Yersinia: novel antivirulence agents. - Wikidata - awgldk - TriplyDB

N-hydroxybenzimidazole inhibitors of the transcription factor LcrF in Yersinia: novel antivirulence agents.

N-hydroxybenzimidazole inhibitors of the transcription factor LcrF in Yersinia: novel antivirulence agents.

http://www.wikidata.org/entity/Q37423363

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Environmental Regulation of Yersinia Pathophysiology Small-Molecule Inhibitors of the Type III Secretion System Chemical inhibitors of the type three secretion system: disarming bacterial pathogens Bioactive heterocycles containing endocyclic N-hydroxy groups Isolation of Salmonella mutants resistant to the inhibitory effect of Salicylidene acylhydrazides on flagella-mediated motility Mechanism of Action and Initial, In Vitro SAR of an Inhibitor of the Shigella flexneri Virulence Regulator VirF Potential novel antibiotics from HTS targeting the virulence-regulating transcription factor, VirF, from Shigella flexneri.Ribavirin suppresses bacterial virulence by targeting LysR-type transcriptional regulators High-throughput screening of the virulence regulator VirF: a novel antibacterial target for shigellosis.MarA, SoxS and Rob of Escherichia coli - Global regulators of multidrug resistance, virulence and stress response.Small molecule inhibitors of LcrF, a Yersinia pseudotuberculosis transcription factor, attenuate virulence and limit infection in a murine pneumonia model.Identification of small-molecule inhibitors of Yersinia pestis Type III secretion system YscN ATPase.The Listeria monocytogenes σB regulon and its virulence-associated functions are inhibited by a small molecule Identification of a small-molecule inhibitor of bacterial AraC family activators.Inhibition of Pseudomonas aeruginosa ExsA DNA-Binding Activity by N-Hydroxybenzimidazoles.Novel agents to inhibit microbial virulence and pathogenicity.Targeting the type III secretion system to treat bacterial infections.Bacterial surface appendages as targets for novel antibacterial therapeutics.Approaches targeting the type III secretion system to treat or prevent bacterial infections.Synthetic Cyclic Peptomers as Type III Secretion System Inhibitors.Anti-virulence Strategies to Target Bacterial Infections.RcsB positively regulates the Yersinia Ysc-Yop type III secretion system by activating expression of the master transcriptional regulator LcrF.Small molecules aimed at type III secretion systems to inhibit bacterial virulence The bacterial type III secretion system as a target for developing new antibiotics

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N-hydroxybenzimidazole inhibitors of the transcription factor LcrF in Yersinia: novel antivirulence agents.

http://www.wikidata.org/entity/Q37423363

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scientific article published on September 2009

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N-hydroxybenzimidazole inhibit ...... a: novel antivirulence agents.

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N-hydroxybenzimidazole inhibit ...... a: novel antivirulence agents.

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N-hydroxybenzimidazole inhibit ...... a: novel antivirulence agents.

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N-hydroxybenzimidazole inhibit ...... a: novel antivirulence agents.

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Journal of Medicinal Chemistry

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N-hydroxybenzimidazole inhibit ...... a: novel antivirulence agents.

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Ann B Macone

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Atul K Verma

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Gabriel Medjanis

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Janice E Donatelli

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Lynne K Garrity-Ryan

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Mark C Grier

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Michael N Alekshun

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Oak K Kim

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S Ken Tanaka

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Stuart B Levy

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P2860

Yersinia pestis, the cause of plague, is a recently emerged clone of Yersinia pseudotuberculosis

Crystal structure of the Escherichia coli Rob transcription factor in complex with DNA

A novel DNA-binding motif in MarA: the first structure for an AraC family transcriptional activator

Biological weapons--a primer for microbiologists

Arac/XylS family of transcriptional regulators

Yersinia pestis--etiologic agent of plague

Targeting type III secretion in Yersinia pestis.

Generation of Yersinia pestis attenuated strains by signature-tagged mutagenesis in search of novel vaccine candidates

Targeting bacterial virulence: inhibitors of type III secretion in Yersinia.

Multiple antimicrobial resistance in plague: an emerging public health risk.

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scholarly article

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10.1021/JM9006577

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2009-09-01T00:00:00Z

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