Advanced glycation end products in extracellular matrix proteins contribute to the failure of sensory nerve regeneration in diabetes.
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The Role of the Reactive Oxygen Species and Oxidative Stress in the Pathomechanism of the Age-Related Ocular Diseases and Other Pathologies of the Anterior and Posterior Eye Segments in AdultsSite-specific AGE modifications in the extracellular matrix: a role for glyoxal in protein damage in diabetesNutrient excess and altered mitochondrial proteome and function contribute to neurodegeneration in diabetesNa+/H+-exchanger-1 inhibition counteracts diabetic cataract formation and retinal oxidative-nitrative stress and apoptosisDiabetic Polyneuropathy in Type 2 Diabetes Mellitus: Inflammation, Oxidative Stress, and Mitochondrial Function.The Impact of Microbiota-Gut-Brain Axis on Diabetic Cognition Impairment.Research on the traditional Chinese medicine treating gastrointestinal motility in diabetic rats by improving biomechanical remodeling and neuroendocrine regulation.Diabetic neuropathy: clinical manifestations and current treatments.AGE metabolites: a biomarker linked to cancer disparity?Ferricytochrome (c) directly oxidizes aminoacetone to methylglyoxal, a catabolite accumulated in carbonyl stress.Klotho depletion contributes to increased inflammation in kidney of the db/db mouse model of diabetes via RelA (serine)536 phosphorylation.Glucose autoxidation induces functional damage to proteins via modification of critical arginine residuesImpaired neurovascular repair in subjects with diabetes following experimental intracutaneous axotomyAdvanced glycation end products and oxidative stress in type 2 diabetes mellitus.Metanx alleviates multiple manifestations of peripheral neuropathy and increases intraepidermal nerve fiber density in Zucker diabetic fatty rats.Diabetic peripheral neuropathy: should a chaperone accompany our therapeutic approach?Site specific modification of the human plasma proteome by methylglyoxal.Glycation of vitronectin inhibits VEGF-induced angiogenesis by uncoupling VEGF receptor-2-αvβ3 integrin cross-talk.Role of Glyoxalase 1 (Glo1) and methylglyoxal (MG) in behavior: recent advances and mechanistic insights.Glycation exacerbates the neuronal toxicity of β-amyloid.Acute carbonyl stress induces occludin glycation and brain microvascular endothelial barrier dysfunction: role for glutathione-dependent metabolism of methylglyoxal.Dicarbonyls and glyoxalase in disease mechanisms and clinical therapeuticsFibronectin matrix mimetics promote full-thickness wound repair in diabetic mice.Small- and large-fiber neuropathy after 40 years of type 1 diabetes: associations with glycemic control and advanced protein glycation: the Oslo Study.In-vivo, non-invasive detection of hyperglycemic states in animal models using mm-wave spectroscopyThe Glyoxalase System and Methylglyoxal-Derived Carbonyl Stress in Sepsis: Glycotoxic Aspects of Sepsis Pathophysiology.Advanced glycation endproducts and their pathogenic roles in neurological disorders.Protein damage in diabetes and uremia--identifying hotspots of proteome damage where minimal modification is amplified to marked pathophysiological effect.Diabetic microvascular complications: possible targets for improved macrovascular outcomesIntegrins and the extracellular matrix: key mediators of development and regeneration of the sensory nervous system.Diabetic neuropathy: cellular mechanisms as therapeutic targets.Proregenerative properties of ECM molecules.Dicarbonyl proteome and genome damage in metabolic and vascular disease.Mechanism of diabetic neuropathy: Where are we now and where to go?Mechanisms of distal axonal degeneration in peripheral neuropathies.Oxidation as an important factor of protein damage: Implications for Maillard reaction.Recent advances in detection of AGEs: Immunochemical, bioanalytical and biochemical approaches.Intracellular Accumulation of Methylglyoxal by Glyoxalase 1 Knock Down Alters Collagen Homoeostasis in L6 Myoblasts.Impaired Axonal Regeneration in Diabetes. Perspective on the Underlying Mechanism from In Vivo and In Vitro Experimental Studies.Morphometric analysis of connective tissue sheaths of sural nerve in diabetic and nondiabetic patients
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Advanced glycation end products in extracellular matrix proteins contribute to the failure of sensory nerve regeneration in diabetes.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 31 August 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Advanced glycation end product ...... erve regeneration in diabetes.
@en
Advanced glycation end product ...... erve regeneration in diabetes.
@nl
type
label
Advanced glycation end product ...... erve regeneration in diabetes.
@en
Advanced glycation end product ...... erve regeneration in diabetes.
@nl
prefLabel
Advanced glycation end product ...... erve regeneration in diabetes.
@en
Advanced glycation end product ...... erve regeneration in diabetes.
@nl
P2093
P2860
P356
P1433
P1476
Advanced glycation end product ...... erve regeneration in diabetes.
@en
P2093
Beatriz Duran-Jimenez
Charles H Streuli
Darin Dobler
David R Tomlinson
Naila Rabbani
Sarah Moffatt
P2860
P304
P356
10.2337/DB09-0320
P407
P577
2009-08-31T00:00:00Z