Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis. - Wikidata - awgldk - TriplyDB

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

http://www.wikidata.org/entity/Q37430683

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ClC-2 regulation of intestinal barrier function: Translation of basic science to therapeutic target Matrix metalloproteinase 9-induced increase in intestinal epithelial tight junction permeability contributes to the severity of experimental DSS colitis Hydrogen sulfide from a NaHS source attenuates dextran sulfate sodium (DSS)-induced inflammation via inhibiting nuclear factor-κB Matrine alleviates lipopolysaccharide-induced intestinal inflammation and oxidative stress via CCR7 signal.Chloride channel ClC- 2 enhances intestinal epithelial tight junction barrier function via regulation of caveolin-1 and caveolar trafficking of occludin.Fluoride: a risk factor for inflammatory bowel disease?Genistein improves inflammatory response and colonic function through NF-κB signal in DSS-induced colonic injury.Pharmaceutical Activation or Genetic Absence of ClC-2 Alters Tight Junctions During Experimental Colitis.

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Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

http://www.wikidata.org/entity/Q37430683

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on December 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@en

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@nl

type

label

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@en

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@nl

prefLabel

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@en

Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis.

@nl

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Inflammatory Bowel Diseases

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Chloride channel ClC-2 is a key factor in the development of DSS-induced murine colitis

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Karen Young

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Meghali Nighot

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Nitsan Maharshak

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Prashant Nighot

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Scott E Plevy

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Thomas Ma

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P2860

Loss of hyperpolarization-activated Cl(-) current in salivary acinar cells from Clcn2 knockout mice

Functional evaluation of human ClC-2 chloride channel mutations associated with idiopathic generalized epilepsies

Male germ cells and photoreceptors, both dependent on close cell-cell interactions, degenerate upon ClC-2 Cl(-) channel disruption

Intestinal mucosal barrier function in health and disease

Caveolin-1-dependent occludin endocytosis is required for TNF-induced tight junction regulation in vivo.

Targeting of tumor necrosis factor receptor 1 to low density plasma membrane domains in human endothelial cells.

Occludin is required for cytokine-induced regulation of tight junction barriers.

A synthetic peptide corresponding to the extracellular domain of occludin perturbs the tight junction permeability barrier.

Coordinated epithelial NHE3 inhibition and barrier dysfunction are required for TNF-mediated diarrhea in vivo.

Amazing chloride channels: an overview.

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2867-2877

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scholarly article

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10.1097/MIB.0B013E3182A82AE9

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13

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19

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Anthony T. Blikslager

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2013-12-01T00:00:00Z

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24030525

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3880115

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