Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia - Wikidata - awgldk - TriplyDB

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

http://www.wikidata.org/entity/Q37430764

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Genetic insights into the mechanisms of Fgf signaling The Role of fibroblast growth factor receptor substrate 2 (FRS2) in the regulation of two activity levels of the components of the extracellular signal-regulated kinase (ERK) pathway in the mouse epididymis.Fgfr1 and the IIIc isoform of Fgfr2 play critical roles in the metanephric mesenchyme mediating early inductive events in kidney development.Role of fibroblast growth factor receptor signaling in kidney development.Genes in the ureteric budding pathway: association study on vesico-ureteral reflux patients.Deletion of fibroblast growth factor receptor 2 from the peri-wolffian duct stroma leads to ureteric induction abnormalities and vesicoureteral reflux Independent roles of Fgfr2 and Frs2alpha in ureteric epithelium.Ablation of the renal stroma defines its critical role in nephron progenitor and vasculature patterning.Fibroblast growth factor receptor-Frs2α signaling is critical for nephron progenitors.Fgfr2 is integral for bladder mesenchyme patterning and function Ureteric morphogenesis requires Fgfr1 and Fgfr2/Frs2α signaling in the metanephric mesenchyme.Fibroblast growth factor receptor signaling in kidney and lower urinary tract development.To bud or not to bud: the RET perspective in CAKUT.Genetics of renal hypoplasia: insights into the mechanisms controlling nephron endowment.Renin-angiotensin system in ureteric bud branching morphogenesis: insights into the mechanisms.Receptor tyrosine kinases in kidney development.Role of FGFRL1 and other FGF signaling proteins in early kidney development.Developmental Genetics and Congenital Anomalies of the Kidney and Urinary Tract.The early development of the kidney and implications for future health.Fgfr2 is integral for bladder mesenchyme patterning and function.

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Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

http://www.wikidata.org/entity/Q37430764

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 09 September 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

@en

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia.

@nl

type

label

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

@en

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia.

@nl

prefLabel

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

@en

Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia.

@nl

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AJPRENAL.00262.2009

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American Journal of Physiology - Renal Physiology

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Deletion of Frs2alpha from the ureteric epithelium causes renal hypoplasia

@en

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Brian Becknell

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Carlton M Bates

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Fen Wang

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Jue Zhang

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Kayle Kish

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Sunder Sims-Lucas

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Multiple fibroblast growth factors support growth of the ureteric bud but have different effects on branching morphogenesis

Docking protein FRS2 links the protein tyrosine kinase RET and its oncogenic forms with the mitogen-activated protein kinase signaling cascade

FRS2 proteins recruit intracellular signaling pathways by binding to diverse targets on fibroblast growth factor and nerve growth factor receptors

Stimulation of phosphatidylinositol 3-kinase by fibroblast growth factor receptors is mediated by coordinated recruitment of multiple docking proteins

Critical role for the docking-protein FRS2 alpha in FGF receptor-mediated signal transduction pathways

Fibroblast growth factors, their receptors and signaling

Identification of SNT/FRS2 docking site on RET receptor tyrosine kinase and its role for signal transduction

Promotion and attenuation of FGF signaling through the Ras-MAPK pathway

Behavioral and neurochemical alterations in mice deficient in anaplastic lymphoma kinase suggest therapeutic potential for psychiatric indications

Role of fibroblast growth factor receptors 1 and 2 in the metanephric mesenchyme

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scholarly article

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10.1152/AJPRENAL.00262.2009

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John F. Bertram

Veraragavan P. Eswarakumar

Luise A Cullen-McEwen

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2009-09-09T00:00:00Z

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