Degradation of amyloid beta protein by purified myelin basic protein. - Wikidata - awgldk - TriplyDB

Degradation of amyloid beta protein by purified myelin basic protein.

Degradation of amyloid beta protein by purified myelin basic protein.

http://www.wikidata.org/entity/Q37431176

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Neurochemical profile of dementia pugilistica Association of Amyloid Pathology With Myelin Alteration in Preclinical Alzheimer Disease.Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer's disease Degradation of soluble and fibrillar amyloid beta-protein by matrix metalloproteinase (MT1-MMP) in vitro Vaccination via Chloroplast Genetics: Affordable Protein Drugs for the Prevention and Treatment of Inherited or Infectious Human Diseases The formation of tau pore-like structures is prevalent and cell specific: possible implications for the disease phenotypes.N-terminal region of myelin basic protein reduces fibrillar amyloid-β deposition in Tg-5xFAD mice.Myelin basic protein associates with AβPP, Aβ1-42, and amyloid plaques in cortex of Alzheimer's disease brain Subjects harboring presenilin familial Alzheimer's disease mutations exhibit diverse white matter biochemistry alterations.Oral delivery of bioencapsulated proteins across blood-brain and blood-retinal barriers.From gut to brain: bioencapsulated therapeutic protein reduces amyloid load upon oral delivery.The multiple roles of myelin protein genes during the development of the oligodendrocyte.Axon-glial disruption: the link between vascular disease and Alzheimer's disease?Are amyloid-degrading enzymes viable therapeutic targets in Alzheimer's disease?Proteolytic degradation of amyloid β-protein.Organotypic vibrosections from whole brain adult Alzheimer mice (overexpressing amyloid-precursor-protein with the Swedish-Dutch-Iowa mutations) as a model to study clearance of beta-amyloid plaques.Myelin basic protein cleaves cell adhesion molecule L1 and promotes neuritogenesis and cell survival Myelin Basic Protein Cleaves Cell Adhesion Molecule L1 and Improves Regeneration After Injury.Inflammation Combined with Ischemia Produces Myelin Injury and Plaque-Like Aggregates of Myelin, Amyloid-β and AβPP in Adult Rat Brain.Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer's Disease Brain: A Review.

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Degradation of amyloid beta protein by purified myelin basic protein.

http://www.wikidata.org/entity/Q37431176

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 19 August 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Degradation of amyloid beta protein by purified myelin basic protein.

@en

Degradation of amyloid beta protein by purified myelin basic protein.

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type

label

Degradation of amyloid beta protein by purified myelin basic protein.

@en

Degradation of amyloid beta protein by purified myelin basic protein.

@nl

prefLabel

Degradation of amyloid beta protein by purified myelin basic protein.

@en

Degradation of amyloid beta protein by purified myelin basic protein.

@nl

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JBC.M109.050856

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Journal of Biological Chemistry

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Degradation of amyloid beta protein by purified myelin basic protein.

@en

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Mahiuddin Ahmed

http://www.w3.org/2001/XMLSchema#string

Mei-Chen Liao

http://www.w3.org/2001/XMLSchema#string

Steven O Smith

http://www.w3.org/2001/XMLSchema#string

William E Van Nostrand

http://www.w3.org/2001/XMLSchema#string

P2860

beta-Amyloid degradation and Alzheimer's disease

Insulin-degrading enzyme regulates extracellular levels of amyloid beta-protein by degradation

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Acyl peptide hydrolase, a serine proteinase isolated from conditioned medium of neuroblastoma cells, degrades the amyloid-beta peptide

Pathways towards and away from Alzheimer's disease

Insulin-degrading enzyme regulates the levels of insulin, amyloid beta-protein, and the beta-amyloid precursor protein intracellular domain in vivo

Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition

Alzheimer's disease beta-amyloid peptide is increased in mice deficient in endothelin-converting enzyme

The isolation, characterization, and lipid-aggregating properties of a citrulline containing myelin basic protein

Angiotensin-converting enzyme degrades Alzheimer amyloid beta-peptide (A beta ); retards A beta aggregation, deposition, fibril formation; and inhibits cytotoxicity.

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28917-28925

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scholarly article

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10.1074/JBC.M109.050856

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42

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284

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2009-08-19T00:00:00Z

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19692707

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2781437

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