Inhibition of RANK expression and osteoclastogenesis by TLRs and IFN-gamma in human osteoclast precursors - Wikidata - awgldk - TriplyDB

Inhibition of RANK expression and osteoclastogenesis by TLRs and IFN-gamma in human osteoclast precursors

Inhibition of RANK expression and osteoclastogenesis by TLRs and IFN-gamma in human osteoclast precursors

http://www.wikidata.org/entity/Q37438518

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The Modulatory Effects of Mesenchymal Stem Cells on Osteoclastogenesis Feedback inhibition of osteoclastogenesis during inflammation by IL-10, M-CSF receptor shedding, and induction of IRF8 Oxysterols and EBI2 promote osteoclast precursor migration to bone surfaces and regulate bone mass homeostasis Modulation of osteoclastogenesis with macrophage M1- and M2-inducing stimuli Activation of Toll‐like receptor 9 inhibits lipopolysaccharide‐induced receptor activator of nuclear factor kappa‐ B ligand expression in rat B lymphocytes.Retinoic acid increases proliferation of human osteoclast progenitors and inhibits RANKL-stimulated osteoclast differentiation by suppressing RANK Elevated suppressor of cytokine signaling-1 (SOCS-1): a mechanism for dysregulated osteoclastogenesis in HIV transgenic rats Regulation of Embryonic and Postnatal Development by the CSF-1 Receptor.Bone erosion in rheumatoid arthritis: mechanisms, diagnosis and treatment.Simultaneous analysis of T helper subsets (Th1, Th2, Th9, Th17, Th22, Tfh, Tr1 and Tregs) markers expression in periapical lesions reveals multiple cytokine clusters accountable for lesions activity and inactivity status.Ligation of TLR5 promotes myeloid cell infiltration and differentiation into mature osteoclasts in rheumatoid arthritis and experimental arthritis.Direct inhibition of human RANK+ osteoclast precursors identifies a homeostatic function of IL-1beta.The relative timing of exposure to phagocytosable particulates and to osteoclastogenic cytokines is critically important in the determination of myeloid cell fate TLR2-dependent modulation of osteoclastogenesis by Porphyromonas gingivalis through differential induction of NFATc1 and NF-kappaB.Osteoimmunology at the nexus of arthritis, osteoporosis, cancer, and infection.Negative regulation of osteoclastogenesis and bone resorption by cytokines and transcriptional repressors.Molecular mechanisms of the biphasic effects of interferon-γ on osteoclastogenesis.Langerhans cells down-regulate inflammation-driven alveolar bone loss.Toll-Like Receptor 2 Stimulation of Osteoblasts Mediates Staphylococcus Aureus Induced Bone Resorption and Osteoclastogenesis through Enhanced RANKL.TRAP-positive osteoclast precursors mediate ROS/NO-dependent bactericidal activity via TLR4 Porphyromonas gingivalis promotes Th17 inducing pathways in chronic periodontitis.Serum amyloid A inhibits RANKL-induced osteoclast formation.The critical role of toll-like receptors--From microbial recognition to autoimmunity: A comprehensive review.IL-1R/TLR2 through MyD88 Divergently Modulates Osteoclastogenesis through Regulation of Nuclear Factor of Activated T Cells c1 (NFATc1) and B Lymphocyte-induced Maturation Protein-1 (Blimp1).Negative regulation of osteoclast precursor differentiation by CD11b and β2 integrin-B-cell lymphoma 6 signaling Osteoclasts-Key Players in Skeletal Health and Disease.Activated γδ T cells inhibit osteoclast differentiation and resorptive activity in vitro.Osteoclastogenesis and arthritis.Role of T cells in ovariectomy induced bone loss--revisited.Alternative pathways of osteoclastogenesis in inflammatory arthritis.Circulating monocytes: an appropriate model for bone-related study.Toll-like receptors-2 and 4 are overexpressed in an experimental model of particle-induced osteolysis.TREM2 in Neurodegenerative Diseases.Abrasive Endoprosthetic Wear Particles Inhibit IFN-γ Secretion in Human Monocytes Via Upregulating TNF-α-Induced miR-29b.TLRs, future potential therapeutic targets for RA.Psoriatic cutaneous inflammation promotes human monocyte differentiation into active osteoclasts, facilitating bone damage.Osteoimmunology: memorandum for rheumatologists.Involvement of redox balance in in vitro osteoclast formation of RAW 264.7 macrophage cells in response to LPS.IFNβ impairs extracellular matrix formation leading to inhibition of mineralization by effects in the early stage of human osteoblast differentiation.Retinoic acid inhibits NFATc1 expression and osteoclast differentiation.

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P2860

Inhibition of RANK expression and osteoclastogenesis by TLRs and IFN-gamma in human osteoclast precursors

http://www.wikidata.org/entity/Q37438518

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Inhibition of RANK expression ...... in human osteoclast precursors

@en

Inhibition of RANK expression ...... n human osteoclast precursors.

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type

label

Inhibition of RANK expression ...... in human osteoclast precursors

@en

Inhibition of RANK expression ...... n human osteoclast precursors.

@nl

prefLabel

Inhibition of RANK expression ...... in human osteoclast precursors

@en

Inhibition of RANK expression ...... n human osteoclast precursors.

@nl

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Journal of Immunology

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Inhibition of RANK expression ...... in human osteoclast precursors

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Jong-Dae Ji

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Kevin P McHugh

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Kyung-Hyun Park-Min

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Lionel B Ivashkiv

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Roberto J Fajardo

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Steven R Goldring

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Zenxin Shen

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P2860

Stimulation by toll-like receptors inhibits osteoclast differentiation

Mutations in two genes encoding different subunits of a receptor signaling complex result in an identical disease phenotype

Synthesis of membrane-bound colony-stimulating factor 1 (CSF-1) and downmodulation of CSF-1 receptors in NIH 3T3 cells transformed by cotransfection of the human CSF-1 and c-fms (CSF-1 receptor) genes

Osteoimmunology: interactions of the bone and immune system

Impaired differentiation of osteoclasts in TREM-2-deficient individuals

Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems

Cytokines in the pathogenesis of rheumatoid arthritis

ADAMs: key components in EGFR signalling and development

Recognition of microorganisms and activation of the immune response

Negative regulation of toll-like receptor-mediated immune responses

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Toll-like receptor

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