Epistatic interactions govern chemically-induced lung tumor susceptibility and Kras mutation site in murine C57BL/6J-ChrA/J chromosome substitution strains.
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Activation of liver X receptor inhibits the development of pulmonary carcinomas induced by 3-methylcholanthrene and butylated hydroxytoluene in BALB/c miceThe Kras mutational spectra of chemically induced lung tumors in different inbred mice mimics the spectra of KRAS mutations in adenocarcinomas in smokers versus nonsmokers.Rare codons capacitate Kras-driven de novo tumorigenesis.The error-prone DNA polymerase ι provides quantitative resistance to lung tumorigenesis and mutagenesis in mice.WeSME: uncovering mutual exclusivity of cancer drivers and beyond.Loss of the SWI/SNF ATPase subunits BRM and BRG1 drives lung cancer development.
P2860
Epistatic interactions govern chemically-induced lung tumor susceptibility and Kras mutation site in murine C57BL/6J-ChrA/J chromosome substitution strains.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Epistatic interactions govern ...... romosome substitution strains.
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Epistatic interactions govern ...... romosome substitution strains.
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Epistatic interactions govern ...... romosome substitution strains.
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Epistatic interactions govern ...... romosome substitution strains.
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Epistatic interactions govern ...... romosome substitution strains.
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Epistatic interactions govern ...... romosome substitution strains.
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P2093
P2860
P356
P1476
Epistatic interactions govern ...... romosome substitution strains.
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P2093
Alvin M Malkinson
Annie Hill-Baskin
Jay McQuillan
Joseph H Nadeau
Lori D Dwyer-Nield
Richard A Radcliffe
P2860
P304
P356
10.1002/IJC.24743
P577
2010-01-01T00:00:00Z