Discharge of RVLM vasomotor neurons is not increased in anesthetized angiotensin II-salt hypertensive rats.
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A Salt-Induced Reno-Cerebral Reflex Activates Renin-Angiotensin Systems and Promotes CKD ProgressionChanges in the inspiratory pattern contribute to modulate the sympathetic activity in sino-aortic denervated rats.Renal responses produced by microinjection of the kappa opioid receptor agonist, U50-488H, into sites within the rat lamina terminalis.Ang II-salt hypertension depends on neuronal activity in the hypothalamic paraventricular nucleus but not on local actions of tumor necrosis factor-α.Characteristics of renal sympathetic nerve single units in rabbits with angiotensin-induced hypertension.
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Discharge of RVLM vasomotor neurons is not increased in anesthetized angiotensin II-salt hypertensive rats.
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scientific article published on 11 October 2013
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Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
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Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
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Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
@en
Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
@nl
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Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
@en
Discharge of RVLM vasomotor ne ...... sin II-salt hypertensive rats.
@nl
P2093
P2860
P1476
Discharge of RVLM vasomotor ne ...... nsin II-salt hypertensive rats
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P2093
Alfredo S Calderon
Glenn M Toney
Mary Ann Andrade
Sergio L Cravo
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P304
P356
10.1152/AJPHEART.00657.2013
P577
2013-10-11T00:00:00Z