A role for epigenetics in the formation of chromosome translocations in acute leukemia. - Wikidata - awgldk - TriplyDB

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

http://www.wikidata.org/entity/Q37441132

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The landscape of DNA methylation-mediated regulation of long non-coding RNAs in breast cancer.

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A role for epigenetics in the formation of chromosome translocations in acute leukemia.

http://www.wikidata.org/entity/Q37441132

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 18 March 2015

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@en

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@nl

type

label

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@en

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@nl

prefLabel

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@en

A role for epigenetics in the formation of chromosome translocations in acute leukemia.

@nl

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J.CANCERGEN.2015.03.006

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Cancer Genetics

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A role for epigenetics in the formation of chromosome translocations in acute leukemia.

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Heidi J Gill Super

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MLL repression domain interacts with histone deacetylases, the polycomb group proteins HPC2 and BMI-1, and the corepressor C-terminal-binding protein

Structure of the MLL CXXC domain-DNA complex and its functional role in MLL-AF9 leukemia.

Rearrangement of the MLL gene in acute lymphoblastic and acute myeloid leukemias with 11q23 chromosomal translocations

The broken MLL gene is frequently located outside the inherent chromosome territory in human lymphoid cells treated with DNA topoisomerase II poison etoposide

MLL targets SET domain methyltransferase activity to Hox gene promoters

Drug-induced histone eviction from open chromatin contributes to the chemotherapeutic effects of doxorubicin.

Intermingling of chromosome territories in interphase suggests role in translocations and transcription-dependent associations

Mediator, TATA-binding protein, and RNA polymerase II contribute to low histone occupancy at active gene promoters in yeast.

The SWR1 histone replacement complex causes genetic instability and genome-wide transcription misregulation in the absence of H2A.Z.

Cancer genetics and epigenetics: two sides of the same coin?

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230-236

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10.1016/J.CANCERGEN.2015.03.006

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