Increased lipid oxidation causes oxidative stress, increased peroxisome proliferator-activated receptor-gamma expression, and diminished pro-osteogenic Wnt signaling in the skeleton.
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Fate decision of mesenchymal stem cells: adipocytes or osteoblasts?Changes in primary lymphoid organs with agingRhoGTPases as key players in mammalian cell adaptation to microgravityThe role of estrogen and androgen receptors in bone health and diseaseStructural Basis for the Association of the Redox-sensitive Target of Rapamycin FATC Domain with Membrane-mimetic MicellesFrom estrogen-centric to aging and oxidative stress: a revised perspective of the pathogenesis of osteoporosisDysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.Association of ALOX15 gene polymorphisms with obesity-related phenotypes in Chinese nuclear families with male offspring.FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in miceLipid peroxidation: production, metabolism, and signaling mechanisms of malondialdehyde and 4-hydroxy-2-nonenalEndogenous glucocorticoids decrease skeletal angiogenesis, vascularity, hydration, and strength in aged mice.Effects of flaxseed lignan and oil on bone health of breast-tumor-bearing mice treated with or without tamoxifen.What old means to bone.T cells: critical bone regulators in health and disease.The immune system and bone.Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.Estrogens and Androgens in Skeletal Physiology and Pathophysiology.Association of Paraoxonase 1 (PON1) polymorphisms with osteoporotic fracture risk in postmenopausal Korean women.The roles of lipid oxidation products and receptor activator of nuclear factor-κB signaling in atherosclerotic calcificationUnraveling the role of FoxOs in bone--insights from mouse models.Marrow fat metabolism is linked to the systemic energy metabolismGlucocorticoids and tumor necrosis factor α increase oxidative stress and suppress Wnt protein signaling in osteoblastsImpact of targeted PPARγ disruption on bone remodeling.The effects of hyperlipidemia on implant osseointegration in the mouse femurAdverse effects of hyperlipidemia on bone regeneration and strength.Hyperlipidemia induces resistance to PTH bone anabolism in mice via oxidized lipids.Cyclophilin D Knock-Out Mice Show Enhanced Resistance to Osteoporosis and to Metabolic Changes Observed in Aging BoneGLUT10 deficiency leads to oxidative stress and non-canonical αvβ3 integrin-mediated TGFβ signalling associated with extracellular matrix disarray in arterial tortuosity syndrome skin fibroblastsThe involvement of the wnt signaling pathway and TCF7L2 in diabetes mellitus: The current understanding, dispute, and perspectiveEvaluation of Glutathione-S-Transferase P1 Polymorphism and its Relation to Bone Mineral Density in Egyptian Children and Adolescents with Beta-Thalassemia MajorEstrogen receptor-α signaling in osteoblast progenitors stimulates cortical bone accrualSelenium in bone health: roles in antioxidant protection and cell proliferation.4-Hydroxynonenal differentially regulates adiponectin gene expression and secretion via activating PPARγ and accelerating ubiquitin-proteasome degradationTanshinol Rescues the Impaired Bone Formation Elicited by Glucocorticoid Involved in KLF15 Pathway.Forum on aging and skeletal health: summary of the proceedings of an ASBMR workshopRegulatory pathways associated with bone loss and bone marrow adiposity caused by aging, chemotherapy, glucocorticoid therapy and radiotherapy.The changing balance between osteoblastogenesis and adipogenesis in aging and its impact on hematopoiesisAging mechanisms in bone.Peroxisome-proliferator-activated receptors regulate redox signaling in the cardiovascular systemFOXOs attenuate bone formation by suppressing Wnt signaling.
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Increased lipid oxidation causes oxidative stress, increased peroxisome proliferator-activated receptor-gamma expression, and diminished pro-osteogenic Wnt signaling in the skeleton.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 05 August 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@en
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@nl
type
label
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@en
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@nl
prefLabel
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@en
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@nl
P2093
P2860
P356
P1476
Increased lipid oxidation caus ...... Wnt signaling in the skeleton.
@en
P2093
Elena Ambrogini
Maria Almeida
Robert L Jilka
Stavros C Manolagas
P2860
P304
27438-27448
P356
10.1074/JBC.M109.023572
P407
P577
2009-08-05T00:00:00Z