Critical role for interleukin-1beta (IL-1beta) during Chlamydia muridarum genital infection and bacterial replication-independent secretion of IL-1beta in mouse macrophages.
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Danger signals, inflammasomes, and the intricate intracellular lives of chlamydiaeChlamydia muridarum Infection of Macrophages Stimulates IL-1β Secretion and Cell Death via Activation of Caspase-1 in an RIP3-Independent MannerChlamydial infection of monocytes stimulates IL-1beta secretion through activation of the NLRP3 inflammasomeRole of extracellular nucleotides in the immune response against intracellular bacteria and protozoan parasitesPlasmid-cured Chlamydia caviae activates TLR2-dependent signaling and retains virulence in the guinea pig model of genital tract infection.Nitric oxide synthases and tubal ectopic pregnancies induced by Chlamydia infection: basic and clinical insights.MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resolConjunctival transcriptome in scarring trachomaThe duration of Chlamydia muridarum genital tract infection and associated chronic pathological changes are reduced in IL-17 knockout mice but protection is not increased further by immunization.The regulation of nitric oxide synthase isoform expression in mouse and human fallopian tubes: potential insights for ectopic pregnancy.Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infectionEnhanced neutrophil longevity and recruitment contribute to the severity of oviduct pathology during Chlamydia muridarum infection.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionPlac8-dependent and inducible NO synthase-dependent mechanisms clear Chlamydia muridarum infections from the genital tractEffect of Mucosal Cytokine Administration on Selective Expansion of Vaginal Dendritic Cells to Support Nanoparticle Transport.Nod1, but not the ASC inflammasome, contributes to induction of IL-1β secretion in human trophoblasts after sensing of Chlamydia trachomatisDetection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Prevention of Chlamydia-induced infertility by inhibition of local caspase activity.Highly heterogeneous, activated, and short-lived regulatory T cells during chronic filarial infection.OT-1 mice display minimal upper genital tract pathology following primary intravaginal Chlamydia muridarum infection.Haemophilus ducreyi infection induces activation of the NLRP3 inflammasome in nonpolarized but not in polarized human macrophages.IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.Clash of the Cytokine Titans: counter-regulation of interleukin-1 and type I interferon-mediated inflammatory responses.Towards a Chlamydia trachomatis vaccine: how close are we?Chlamydiales and the innate immune response: friend or foe?A Macaque Model for Rectal Lymphogranuloma Venereum and Non-Lymphogranuloma Venereum Chlamydia trachomatis: Impact on Rectal Simian/Human Immunodeficiency Virus Acquisition.Development of a rectal sexually transmitted infection (STI) Model in Rhesus macaques using Chlamydia trachomatis serovars E and L2.Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Immunopathogenesis of Chlamydial Infections.Murine MicroRNA-214 regulates intracellular adhesion molecule (ICAM1) gene expression in genital Chlamydia muridarum infection.Immunity against a Chlamydia infection and disease may be determined by a balance of IL-17 signaling.Chlamydia muridarum major outer membrane protein-specific antibodies inhibit in vitro infection but enhance pathology in vivo.Whole-Exome Sequencing to Identify Novel Biological Pathways Associated With Infertility After Pelvic Inflammatory Disease.B cell presentation of Chlamydia antigen selects out protective CD4γ13 T cells; implications for genital tract tissue resident memory lymphocyte clusters.Differential signaling pathways are initiated in macrophages during infection depending on the intracellular fate of Chlamydia spp.Discovery of Blood Transcriptional Endotypes in Women with Pelvic Inflammatory Disease.Toll-like receptor 3 (TLR3) promotes the resolution of Chlamydia muridarum genital tract infection in congenic C57BL/6N mice.Molecular mechanisms of tubal pregnancy
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P2860
Critical role for interleukin-1beta (IL-1beta) during Chlamydia muridarum genital infection and bacterial replication-independent secretion of IL-1beta in mouse macrophages.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 05 October 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Critical role for interleukin- ...... IL-1beta in mouse macrophages.
@en
Critical role for interleukin-1beta
@nl
type
label
Critical role for interleukin- ...... IL-1beta in mouse macrophages.
@en
Critical role for interleukin-1beta
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prefLabel
Critical role for interleukin- ...... IL-1beta in mouse macrophages.
@en
Critical role for interleukin-1beta
@nl
P2093
P2860
P356
P1476
Critical role for interleukin- ...... IL-1beta in mouse macrophages.
@en
P2093
Charles W Andrews
Daniel Prantner
Helmut Brade
James D Sikes
Roger G Rank
Toni Darville
Uma M Nagarajan
P2860
P304
P356
10.1128/IAI.00883-09
P407
P577
2009-10-05T00:00:00Z