IFNγ/IL-10 co-producing cells dominate the CD4 response to malaria in highly exposed children.
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Expanding the antimalarial toolkit: Targeting host-parasite interactionsImmunoregulation in human malaria: the challenge of understanding asymptomatic infectionInterferon-γ: The Jekyll and Hyde of MalariaCD4 T-cell subsets in malaria: TH1/TH2 revisitedThe Regulation of CD4(+) T Cell Responses during Protozoan InfectionsAcute Malaria Induces PD1+CTLA4+ Effector T Cells with Cell-Extrinsic Suppressor FunctionCombined Immune Therapy for the Treatment of Visceral LeishmaniasisType I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental MalariaImpact of In Utero Exposure to Malaria on Fetal T Cell Immunity.CD4 T-cell expression of IFN-γ and IL-17 in pediatric malarial anemia.Resisting and tolerating P. falciparum in pregnancy under different malaria transmission intensitiesLoss and dysfunction of Vδ2⁺ γδ T cells are associated with clinical tolerance to malaria.Exposure-dependent control of malaria-induced inflammation in children.Decline of FoxP3+ Regulatory CD4 T Cells in Peripheral Blood of Children Heavily Exposed to MalariaBlimp-1-Dependent IL-10 Production by Tr1 Cells Regulates TNF-Mediated Tissue Pathology.Novel serologic biomarkers provide accurate estimates of recent Plasmodium falciparum exposure for individuals and communitiesEffector Phenotype of Plasmodium falciparum-Specific CD4+ T Cells Is Influenced by Both Age and Transmission Intensity in Naturally Exposed PopulationsBiosignatures of Exposure/Transmission and ImmunityIL-27 and TGFβ mediated expansion of Th1 and adaptive regulatory T cells expressing IL-10 correlates with bacterial burden and disease severity in pulmonary tuberculosis.A Novel Model of Asymptomatic Plasmodium Parasitemia That Recapitulates Elements of the Human Immune Response to Chronic Infection.The effect of declining exposure on T cell-mediated immunity to Plasmodium falciparum - an epidemiological "natural experiment".Macrophage Colony Stimulating Factor Derived from CD4+ T Cells Contributes to Control of a Blood-Borne InfectionT cell subtypes and reciprocal inflammatory mediator expression differentiate P. falciparum memory recall responses in asymptomatic and symptomatic malaria patients in southeastern Haiti.B cell sub-types following acute malaria and associations with clinical immunity.Activated Neutrophils Are Associated with Pediatric Cerebral Malaria Vasculopathy in Malawian ChildrenIFNγ Responses to Pre-erythrocytic and Blood-stage Malaria Antigens Exhibit Differential Associations With Past Exposure and Subsequent Protection.Delayed acquisition of Plasmodium falciparum antigen-specific CD4(+) T cell responses in HIV-exposed uninfected Malawian children receiving daily cotrimoxazole prophylaxisEffective Antimalarial Chemoprevention in Childhood Enhances the Quality of CD4+ T Cells and Limits Their Production of Immunoregulatory Interleukin 10.Transcriptomic evidence for modulation of host inflammatory responses during febrile Plasmodium falciparum malaria.Type I Interferons Regulate Immune Responses in Humans with Blood-Stage Plasmodium falciparum Infection.NK Cells: Uncertain Allies against Malaria.The immunological balance between host and parasite in malaria.Maternal Microchimerism Predicts Increased Infection but Decreased Disease due to Plasmodium falciparum During Early Childhood.Quantifying Heterogeneous Malaria Exposure and Clinical Protection in a Cohort of Ugandan Children.The Impact of Established Immunoregulatory Networks on Vaccine Efficacy and the Development of Immunity to Malaria.Cutting Edge: IL-10 Is Essential for the Generation of Germinal Center B Cell Responses and Anti-Plasmodium Humoral Immunity.Long-Lived CD4+IFN-γ+ T Cells rather than Short-Lived CD4+IFN-γ+IL-10+ T Cells Initiate Rapid IL-10 Production To Suppress Anamnestic T Cell Responses during Secondary Malaria Infection.Parasite-Specific CD4+ IFN-γ+ IL-10+ T Cells Distribute within Both Lymphoid and Nonlymphoid Compartments and Are Controlled Systemically by Interleukin-27 and ICOS during Blood-Stage Malaria InfectionThe Development of Plasmodium falciparum-Specific IL10 CD4 T Cells and Protection from Malaria in Children in an Area of High Malaria Transmission.Evaluating anti-disease immunity to malaria and implications for vaccine design.
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IFNγ/IL-10 co-producing cells dominate the CD4 response to malaria in highly exposed children.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 09 January 2014
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@en
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@nl
type
label
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@en
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@nl
prefLabel
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@en
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@nl
P2093
P2860
P50
P1433
P1476
IFNγ/IL-10 co-producing cells ...... ia in highly exposed children.
@en
P2093
Charles Ebusu
Felistas Nankya
Ijeoma Eccles-James
Jessica Briggs
Katherine Bowen
Margaret E Feeney
Mary K Muhindo
Prasanna Jagannathan
Samuel Wamala
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003864
P577
2014-01-09T00:00:00Z