Nrf2 protects against TWEAK-mediated skeletal muscle wasting. - Wikidata - awgldk - TriplyDB

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

http://www.wikidata.org/entity/Q37459975

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The role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymes Disruption of nuclear factor (erythroid-derived-2)-like 2 antioxidant signaling: a mechanism for impaired activation of stem cells and delayed regeneration of skeletal muscle Tumor Necrosis Factor Alpha and Insulin-Like Growth Factor 1 Induced Modifications of the Gene Expression Kinetics of Differentiating Skeletal Muscle Cells.Role of Nrf2 Signaling in the Regulation of Vascular BK Channel β1 Subunit Expression and BK Channel Function in High-Fat Diet-Induced Diabetic Mice.Partial involvement of Nrf2 in skeletal muscle mitohormesis as an adaptive response to mitochondrial uncoupling.Melatonin prevents mitochondrial dysfunctions and death in differentiated skeletal muscle cells.Role of Keap1-Nrf2 Signaling in Anhedonia Symptoms in a Rat Model of Chronic Neuropathic Pain: Improvement With Sulforaphane

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Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

http://www.wikidata.org/entity/Q37459975

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 10 January 2014

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@en

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@nl

type

label

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@en

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@nl

prefLabel

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@en

Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@nl

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Scientific Reports

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Nrf2 protects against TWEAK-mediated skeletal muscle wasting.

@en

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Christian Rosen

http://www.w3.org/2001/XMLSchema#string

Christoph Jan Wruck

http://www.w3.org/2001/XMLSchema#string

Othman Al-Sawaf

http://www.w3.org/2001/XMLSchema#string

Thomas Pufe

http://www.w3.org/2001/XMLSchema#string

Tolga Taha Sönmez

http://www.w3.org/2001/XMLSchema#string

Yuet Wai Kan

http://www.w3.org/2001/XMLSchema#string

P2860

A novel laser-Doppler flowmetry assisted murine model of acute hindlimb ischemia-reperfusion for free flap research

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

When NRF2 talks, who's listening?

NRF2, a member of the NFE2 family of transcription factors, is not essential for murine erythropoiesis, growth, and development

The TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.

TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.

TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokine

Genetic ablation of TWEAK augments regeneration and post-injury growth of skeletal muscle in mice.

Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.

p53 suppresses the Nrf2-dependent transcription of antioxidant response genes.

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3625

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scholarly article

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10.1038/SREP03625

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4

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Athanassios Fragoulis

Othman Al-Sawaf

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2014-01-10T00:00:00Z

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259651341

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24406502

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3887379

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