Pathway-specific alteration of synaptic plasticity in Tg2576 mice. - Wikidata - awgldk - TriplyDB

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

http://www.wikidata.org/entity/Q37461280

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Spines, plasticity, and cognition in Alzheimer's model mice Dopamine neuronal loss contributes to memory and reward dysfunction in a model of Alzheimer's disease.c-Jun N-terminal kinase has a key role in Alzheimer disease synaptic dysfunction in vivo.Neuritin can normalize neural deficits of Alzheimer's disease.In vivo detection of oxidation-specific epitopes in atherosclerotic lesions using biocompatible manganese molecular magnetic imaging probes Neural stem cells isolated from amyloid precursor protein-mutated mice for drug discovery Astrocytes and synaptic plasticity in health and disease.Age-related synaptic dysfunction in Tg2576 mice starts as a failure in early long-term potentiation which develops into a full abolishment of late long-term potentiation.Accumulation of murine amyloid-β mimics early Alzheimer's disease.An Intranasal Formulation of Erythropoietin (Neuro-EPO) Prevents Memory Deficits and Amyloid Toxicity in the APPSwe Transgenic Mouse Model of Alzheimer's Disease.

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Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

http://www.wikidata.org/entity/Q37461280

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on June 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@en

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@nl

type

label

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@en

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@nl

prefLabel

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@en

Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@nl

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S10059-011-0077-8

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Molecules and Cells

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Pathway-specific alteration of synaptic plasticity in Tg2576 mice.

@en

P2093

Hye-sun Kim

http://www.w3.org/2001/XMLSchema#string

Joung-Hun Kim

http://www.w3.org/2001/XMLSchema#string

Jung Hoon Jung

http://www.w3.org/2001/XMLSchema#string

Kyongman An

http://www.w3.org/2001/XMLSchema#string

Oh Bin Kwon

http://www.w3.org/2001/XMLSchema#string

P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Synaptic plasticity in animal models of early Alzheimer's disease

Synaptic transmission block by presynaptic injection of oligomeric amyloid beta

Regulation of NMDA receptor trafficking by amyloid-beta

Loss of presenilin function causes impairments of memory and synaptic plasticity followed by age-dependent neurodegeneration

Mossy fiber long-term potentiation deficits in BACE1 knock-outs can be rescued by activation of alpha7 nicotinic acetylcholine receptors

Matrix metalloproteinase-9 is required for hippocampal late-phase long-term potentiation and memory.

Age-dependent changes in brain, CSF, and plasma amyloid (beta) protein in the Tg2576 transgenic mouse model of Alzheimer's disease.

Biomarkers for early detection of Alzheimer disease.

Early-onset behavioral and synaptic deficits in a mouse model of Alzheimer's disease.

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s10059-011-0077-8

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197-201

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scholarly article

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10.1007/S10059-011-0077-8

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2

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32

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2011-06-01T00:00:00Z

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21638202

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3887667

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