Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
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Cellular and molecular biology of small cell lung cancer: an overviewTrial Watch:: Oncolytic viruses for cancer therapyOncolytic Seneca Valley Virus: past perspectives and future directionsOncolytic viruses: From bench to bedside with a focus on safety.Oncolytic polio virotherapy of cancerA DLL3-targeted antibody-drug conjugate eradicates high-grade pulmonary neuroendocrine tumor-initiating cells in vivoIntratumoral anti-HuD immunotoxin therapy for small cell lung cancer and neuroblastoma.Seneca Valley Virus 3Cpro Substrate Optimization Yields Efficient Substrates for Use in Peptide-Prodrug TherapyRapamycin rescues ABT-737 efficacy in small cell lung cancerDNA methylation in small cell lung cancer defines distinct disease subtypes and correlates with high expression of EZH2.Small Cell Lung Cancer: Can Recent Advances in Biology and Molecular Biology Be Translated into Improved Outcomes?YAP and TAZ modulate cell phenotype in a subset of small cell lung cancer.Seneca Valley Virus Suppresses Host Type I Interferon Production by Targeting Adaptor Proteins MAVS, TRIF, and TANK for Cleavage.Hypermethylation of adjacent CpG sites is negatively correlated with the expression of lineage oncogene ASCL1 in pulmonary neuroendocrine tumors.Unravelling the biology of SCLC: implications for therapy.MYC Drives Progression of Small Cell Lung Cancer to a Variant Neuroendocrine Subtype with Vulnerability to Aurora Kinase Inhibition.Novel oncolytic viral therapies in patients with thoracic malignancies.Family matters: How MYC family oncogenes impact small cell lung cancer.Anthrax toxin receptor 1 is the cellular receptor for Seneca Valley virus.ASCL1 and NEUROD1 Reveal Heterogeneity in Pulmonary Neuroendocrine Tumors and Regulate Distinct Genetic Programs.Panorama from the oncolytic virotherapy summit.Protein expression of TTF1 and cMYC define distinct molecular subgroups of small cell lung cancer with unique vulnerabilities to aurora kinase inhibition, DLL3 targeting, and other targeted therapies.Cryo-EM Structure of Seneca Valley Virus Procapsid.Small-cell lung cancer: what we know, what we need to know and the path forward.Small cell lung cancer tumors and preclinical models display heterogeneity of neuroendocrine phenotypes.Seneca Valley Virus Exploits TEM8, a Collagen Receptor Implicated in Tumor Growth
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Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
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article científic
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article scientifique
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artigo científico
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bilimsel makale
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scientific article published on 05 June 2013
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Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
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Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
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Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
@en
Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
@nl
prefLabel
Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
@en
Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer.
@nl
P2093
P2860
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P1476
Selective tropism of Seneca Valley virus for variant subtype small cell lung cancer
@en
P2093
Christine L Hann
Craig D Peacock
Irina Dobromilskaya
Whei F Moriarty
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P304
P356
10.1093/JNCI/DJT130
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P577
2013-06-05T00:00:00Z