Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
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Oxidative stress during acetaminophen hepatotoxicity: Sources, pathophysiological role and therapeutic potentialc-Jun N-terminal kinase modulates oxidant stress and peroxynitrite formation independent of inducible nitric oxide synthase in acetaminophen hepatotoxicity.The oxygen tension modulates acetaminophen-induced mitochondrial oxidant stress and cell injury in cultured hepatocytesCurrent issues with acetaminophen hepatotoxicity--a clinically relevant model to test the efficacy of natural products.Polyinosinic-polycytidylic acid suppresses acetaminophen-induced hepatotoxicity independent of type I interferons and toll-like receptor 3Schisandrol B protects against acetaminophen-induced hepatotoxicity by inhibition of CYP-mediated bioactivation and regulation of liver regeneration.Acute ethanol causes hepatic mitochondrial depolarization in mice: role of ethanol metabolism.Hepatitis C virus structural proteins can exacerbate or ameliorate acetaminophen-induced liver injury in mice.Resveratrol prevents protein nitration and release of endonucleases from mitochondria during acetaminophen hepatotoxicity.Chlorogenic acid prevents acetaminophen-induced liver injury: the involvement of CYP450 metabolic enzymes and some antioxidant signalsAcetaminophen: Dose-Dependent Drug Hepatotoxicity and Acute Liver Failure in Patients.An indole derivative protects against acetaminophen-induced liver injury by directly binding to N-acetyl-p-benzoquinone imine in mice.Fas receptor-deficient lpr mice are protected against acetaminophen hepatotoxicity due to higher glutathione synthesis and enhanced detoxification of oxidant stress.Protection against acetaminophen-induced liver injury by allopurinol is dependent on aldehyde oxidase-mediated liver preconditioningProtective effect of metallothionein on oxidative stress-induced DNA damage.Pharmacokinetic variations in cancer patients with liver dysfunction: applications and challenges of pharmacometabolomics.Macrophage-derived IL-1α promotes sterile inflammation in a mouse model of acetaminophen hepatotoxicity.Kampo formula "Hochu-ekki-to" suppressed carbon tetrachloride-induced hepatotoxicity in mice.In vitro to in vivo extrapolation and species response comparisons for drug-induced liver injury (DILI) using DILIsym™: a mechanistic, mathematical model of DILI.
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Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 October 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@en
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
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type
label
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@en
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@nl
prefLabel
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@en
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@nl
P2093
P2860
P1476
Mechanism of protection by metallothionein against acetaminophen hepatotoxicity.
@en
P2093
Antonio Artigues
Anwar Farhood
Chieko Saito
Hartmut Jaeschke
Hui-Min Yan
Maria T Villar
P2860
P304
P356
10.1016/J.TAAP.2009.10.006
P577
2009-10-14T00:00:00Z