Does inflammation after stroke affect the developing brain differently than adult brain? - Wikidata - awgldk - TriplyDB

Does inflammation after stroke affect the developing brain differently than adult brain?

Does inflammation after stroke affect the developing brain differently than adult brain?

http://www.wikidata.org/entity/Q37466730

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Mechanisms of perinatal arterial ischemic stroke Barrier mechanisms in neonatal stroke Oligodendroglial alterations and the role of microglia in white matter injury: relevance to schizophrenia.Brain development in rodents and humans: Identifying benchmarks of maturation and vulnerability to injury across species.Suppression of microglia activation after hypoxia-ischemia results in age-dependent improvements in neurologic injury.Protection by vascular prostaglandin E2 signaling in hypoxic-ischemic encephalopathy Neurocritical care for neonates.Effect of transient maternal hypotension on apoptotic cell death in foetal rat brain.Prenatal hypoxic-ischemic insult changes the distribution and number of NADPH-diaphorase cells in the cerebellum.Developmental regulation of the neuroinflammatory responses to LPS and/or hypoxia-ischemia between preterm and term neonates: An experimental study.Blood-brain barrier dysfunction in disorders of the developing brain Hypothermia modulates cytokine responses after neonatal rat hypoxic-ischemic injury and reduces brain damage.Molecular and cellular pathways as a target of therapeutic hypothermia: pharmacological aspect Short and long-term analysis and comparison of neurodegeneration and inflammatory cell response in the ipsilateral and contralateral hemisphere of the neonatal mouse brain after hypoxia/ischemia.Microarray Profiling and Co-Expression Network Analysis of LncRNAs and mRNAs in Neonatal Rats Following Hypoxic-ischemic Brain Damage Reduced infarct size and accumulation of microglia in rats treated with WIN 55,212-2 after neonatal stroke.The role of inflammation in perinatal brain injury.Opposite effect of inflammation on subventricular zone versus hippocampal precursors in brain injury Cellular mechanisms of neurovascular damage and repair after stroke PPARγ-induced upregulation of CD36 enhances hematoma resolution and attenuates long-term neurological deficits after germinal matrix hemorrhage in neonatal rats.Genetic deletion of CD36 enhances injury after acute neonatal stroke Blood-brain barrier permeability is increased after acute adult stroke but not neonatal stroke in the rat.Selective anesthesia-induced neuroinflammation in developing mouse brain and cognitive impairment Sevoflurane anesthesia in pregnant mice induces neurotoxicity in fetal and offspring mice.Dexamethasone and betamethasone protect against lipopolysaccharide-induced brain damage in neonatal rats.Acute and chronic vascular responses to experimental focal arterial stroke in the neonate rat.Isoflurane and sevoflurane increase interleukin-6 levels through the nuclear factor-kappa B pathway in neuroglioma cells Inhibition of CXCL12 signaling attenuates the postischemic immune response and improves functional recovery after stroke.Umbilical cord blood therapy potentiated with erythropoietin for children with cerebral palsy: a double-blind, randomized, placebo-controlled trial The choroid plexus as a site of damage in hemorrhagic and ischemic stroke and its role in responding to injury.Pathophysiological role of inflammatory molecules in paediatric ischaemic brain injury.Central nervous system injury and neurobiobehavioral function in children with brain tumors: a review of the literature.Using hormetic strategies to improve ischemic preconditioning and postconditioning against stroke.Long-lasting paracrine effects of human cord blood cells on damaged neocortex in an animal model of cerebral palsy.Perinatal hypoxia-ischemia reduces α 7 nicotinic receptor expression and selective α 7 nicotinic receptor stimulation suppresses inflammation and promotes microglial Mox phenotype Differential effects of minocycline on microglial activation and neurodegeneration following closed head injury in the neonate rat.Prenatal Systemic Hypoxia-Ischemia and Oligodendroglia Loss in Cerebellum.HMGB1 Translocation After Ischemia in the Ovine Fetal Brain.Attenuation of reactive gliosis does not affect infarct volume in neonatal hypoxic-ischemic brain injury in mice.Lack of the scavenger receptor CD36 alters microglial phenotypes after neonatal stroke

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Does inflammation after stroke affect the developing brain differently than adult brain?

http://www.wikidata.org/entity/Q37466730

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 August 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Does inflammation after stroke affect the developing brain differently than adult brain?

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Developmental Neuroscience

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Does inflammation after stroke affect the developing brain differently than adult brain?

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Midori A Yenari

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Zinaida S Vexler

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P2860

Peak plasma interleukin-6 and other peripheral markers of inflammation in the first week of ischaemic stroke correlate with brain infarct volume, stroke severity and long-term outcome

The neuroblast and angioblast chemotaxic factor SDF-1 (CXCL12) expression is briefly up regulated by reactive astrocytes in brain following neonatal hypoxic-ischemic injury.

Chemokines and disease

IKK mediates ischemia-induced neuronal death

Brain inflammation and adult neurogenesis: the dual role of microglia

Immune cells contribute to the maintenance of neurogenesis and spatial learning abilities in adulthood

Interleukin-18 involvement in hypoxic-ischemic brain injury

Erythropoietin attenuates hyperoxia-induced cell death by modulation of inflammatory mediators and matrix metalloproteinases

Anti-inflammatory effects of the 70 kDa heat shock protein in experimental stroke

Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis

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378-393

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10.1159/000232556

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2009-08-11T00:00:00Z

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