IPS-1 signaling has a nonredundant role in mediating antiviral responses and the clearance of respiratory syncytial virus.
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The Absence of Interferon-β Promotor Stimulator-1 (IPS-1) Predisposes to Bronchiolitis and Asthma-like Pathology in Response to Pneumoviral Infection in MiceInnate immune recognition of respiratory syncytial virus infection.Respiratory syncytial virus induced type I IFN production by pDC is regulated by RSV-infected airway epithelial cells, RSV-exposed monocytes and virus specific antibodiesAssessment of genetic associations between common single nucleotide polymorphisms in RIG-I-like receptor and IL-4 signaling genes and severe respiratory syncytial virus infection in children: a candidate gene case-control study.Alveolar macrophage-derived type I interferons orchestrate innate immunity to RSV through recruitment of antiviral monocytesT cell responses are elicited against Respiratory Syncytial Virus in the absence of signalling through TLRs, RLRs and IL-1R/IL-18R.Respiratory syncytial virus infection: an innate perspective.Alveolar Macrophages Can Control Respiratory Syncytial Virus Infection in the Absence of Type I Interferons.Host-Viral Interactions: Role of Pattern Recognition Receptors (PRRs) in Human Pneumovirus InfectionsContribution of autophagy to antiviral immunity.Transient Depletion of CD169+ Cells Contributes to Impaired Early Protection and Effector CD8+ T Cell Recruitment against Mucosal Respiratory Syncytial Virus Infection.IL-27R-mediated regulation of IL-17 controls the development of respiratory syncytial virus-associated pathogenesis.Pulmonary Dendritic Cell Subsets Shape the Respiratory Syncytial Virus-Specific CD8+ T Cell Immunodominance Hierarchy in Neonates.The innate immune response to RSV: Advances in our understanding of critical viral and host factors.Pathogen recognition receptor crosstalk in respiratory syncytial virus sensing: a host and cell type perspective.Respiratory Syncytial Virus Infection Upregulates NLRC5 and Major Histocompatibility Complex Class I Expression through RIG-I Induction in Airway Epithelial Cells.DNGR-1 is dispensable for CD8+ T-cell priming during respiratory syncytial virus infection.Pulmonary Susceptibility of Neonates to Respiratory Syncytial Virus Infection: A Problem of Innate Immunity?
P2860
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P2860
IPS-1 signaling has a nonredundant role in mediating antiviral responses and the clearance of respiratory syncytial virus.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@en
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@nl
type
label
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@en
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@nl
prefLabel
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@en
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@nl
P2093
P2860
P356
P1476
IPS-1 signaling has a nonredun ...... f respiratory syncytial virus.
@en
P2093
Bryan C Petersen
Catherine Ptaschinski
Denise E De Almeida Nagata
Matthew A Schaller
Nicholas W Lukacs
Steven L Kunkel
Sumanta Mukherjee
Susan Morris
Taro Kawai
Tine Demoor
P2860
P304
P356
10.4049/JIMMUNOL.1201763
P407
P50
P577
2012-11-07T00:00:00Z