The matricellular protein periostin is required for sost inhibition and the anabolic response to mechanical loading and physical activity - Wikidata - awgldk - TriplyDB

The matricellular protein periostin is required for sost inhibition and the anabolic response to mechanical loading and physical activity

The matricellular protein periostin is required for sost inhibition and the anabolic response to mechanical loading and physical activity

http://www.wikidata.org/entity/Q37467754

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Sost down-regulation by mechanical strain in human osteoblastic cells involves PGE2 signaling via EP4 Role of Periostin in Adhesion and Migration of Bone Remodeling Cells Novel biological markers of bone: from bone metabolism to bone physiology Parathyroid hormone's enhancement of bones' osteogenic response to loading is affected by ageing in a dose- and time-dependent manner.Identification of Osteoporosis-Associated Protein Biomarkers from Ovariectomized Rat Urine The role of periostin in tissue remodeling across health and disease.Impaired musculoskeletal response to age and exercise in PPARβ(-/-) diabetic mice Mechanical loading-related bone gain is enhanced by tamoxifen but unaffected by fulvestrant in female mice Zoledronate effects on systemic and jaw osteopenias in ovariectomized periostin-deficient mice Periostin in fibrillogenesis for tissue regeneration: periostin actions inside and outside the cell Age-related impairment of bones' adaptive response to loading in mice is associated with sex-related deficiencies in osteoblasts but no change in osteocytes.Diet and gene interactions influence the skeletal response to polyunsaturated fatty acids A method for isolating high quality RNA from mouse cortical and cancellous bone.Periostin deficiency increases bone damage and impairs injury response to fatigue loading in adult mice Risedronate does not reduce mechanical loading-related increases in cortical and trabecular bone mass in mice Periostin responds to mechanical stress and tension by activating the MTOR signaling pathway.The sclerostin-independent bone anabolic activity of intermittent PTH treatment is mediated by T-cell-produced Wnt10b A genome-wide association study of copy-number variation identifies putative loci associated with osteoarthritis in Koreans.An irradiation-altered bone marrow microenvironment impacts anabolic actions of PTH.Biomineralization of bone: a fresh view of the roles of non-collagenous proteins.High-throughput screening of mouse gene knockouts identifies established and novel skeletal phenotypes.Removal of SOST or blocking its product sclerostin rescues defects in the periodontitis mouse model.Mechanical loading-related changes in osteocyte sclerostin expression in mice are more closely associated with the subsequent osteogenic response than the peak strains engendered.The Wnt Inhibitor Sclerostin Is Up-regulated by Mechanical Unloading in Osteocytes in Vitro.Do Non-collagenous Proteins Affect Skeletal Mechanical Properties?Association of CDX1 binding site of periostin gene with bone mineral density and vertebral fracture risk.The role of novel and known extracellular matrix and adhesion molecules in the homeostatic and regenerative bone marrow microenvironment A botulinum toxin-derived targeted secretion inhibitor downregulates the GH/IGF1 axis Regulation of beta catenin signaling and parathyroid hormone anabolic effects in bone by the matricellular protein periostin Preclinical mouse models for assessing axial compression of long bones during exercise.Cortical and trabecular bone adaptation to incremental load magnitudes using the mouse tibial axial compression loading model.Periostin-deficient mice, a relevant animal model to investigate periodontitis or not?Transcriptional profiling of cortical versus cancellous bone from mechanically-loaded murine tibiae reveals differential gene expression.High levels of periostin correlate with increased fracture rate, diffuse MRI pattern, abnormal bone remodeling and advanced disease stage in patients with newly diagnosed symptomatic multiple myeloma.Sclerostin's role in bone's adaptive response to mechanical loading.Adaptive and Injury Response of Bone to Mechanical Loading.Talking among ourselves: paracrine control of bone formation within the osteoblast lineage.Implications of exercise-induced adipo-myokines in bone metabolism.Role and mechanism of action of sclerostin in bone.Cathepsin K Controls Cortical Bone Formation by Degrading Periostin.

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The matricellular protein periostin is required for sost inhibition and the anabolic response to mechanical loading and physical activity

http://www.wikidata.org/entity/Q37467754

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on December 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

The matricellular protein peri ...... loading and physical activity

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The matricellular protein peri ...... loading and physical activity.

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type

label

The matricellular protein peri ...... loading and physical activity

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The matricellular protein peri ...... loading and physical activity.

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prefLabel

The matricellular protein peri ...... loading and physical activity

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The matricellular protein peri ...... loading and physical activity.

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JBC.M109.060335

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Journal of Biological Chemistry

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The matricellular protein peri ...... loading and physical activity

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Estelle N Bianchi

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Kara N Standley

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Michelangelo Foti

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Nicolas Bonnet

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Serge L Ferrari

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Simon J Conway

http://www.w3.org/2001/XMLSchema#string

Vincent Stadelmann

http://www.w3.org/2001/XMLSchema#string

P2860

Periostin secreted by epithelial ovarian carcinoma is a ligand for alpha(V)beta(3) and alpha(V)beta(5) integrins and promotes cell motility

Osteoblast-specific factor 2: cloning of a putative bone adhesion protein with homology with the insect protein fasciclin I

AMPK and PPARdelta agonists are exercise mimetics

The Wnt co-receptor LRP5 is essential for skeletal mechanotransduction but not for the anabolic bone response to parathyroid hormone treatment

LDL receptor-related proteins 5 and 6 in Wnt/beta-catenin signaling: arrows point the way

Mechanical stimulation of bone in vivo reduces osteocyte expression of Sost/sclerostin

Sclerostin antibody treatment increases bone formation, bone mass, and bone strength in a rat model of postmenopausal osteoporosis

Identification and characterization of a novel protein, periostin, with restricted expression to periosteum and periodontal ligament and increased expression by transforming growth factor beta

Molecular profile of catabolic versus anabolic treatment regimens of parathyroid hormone (PTH) in rat bone: an analysis by DNA microarray

Periostin-like-factor in osteogenesis

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35939-35950

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scholarly article

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10.1074/JBC.M109.060335

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P433

51

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284

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2009-12-01T00:00:00Z

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19837663

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2791022

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