PKCδ null mutations in a mouse model of osteoarthritis alter osteoarthritic pain independently of joint pathology by augmenting NGF/TrkA-induced axonal outgrowth.
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Osteoarthritis: toward a comprehensive understanding of pathological mechanism.Nerve growth factor blockade for the management of osteoarthritis pain: what can we learn from clinical trials and preclinical models?Intraarticular slow-release triamcinolone acetate reduces allodynia in an experimental mouse knee osteoarthritis modelNerve growth factor inhibitor with novel-binding domain demonstrates nanomolar efficacy in both cell-based and cell-free assay systems.Traumatic osteoarthritis-induced persistent mechanical hyperalgesia in a rat model of anterior cruciate ligament transection plus a medial meniscectomy.
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PKCδ null mutations in a mouse model of osteoarthritis alter osteoarthritic pain independently of joint pathology by augmenting NGF/TrkA-induced axonal outgrowth.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 January 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
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PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
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PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
@en
PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
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PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
@en
PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
@nl
P2093
P2860
P1476
PKCδ null mutations in a mouse ...... TrkA-induced axonal outgrowth.
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P2093
Andre J van Wijnen
Brett Levine
Daniel A Shelly
Dovrat Brass
Guozhi Xiao
Hee-Jeong Im
Jeffrey S Kroin
Jinyuan Li
P2860
P304
P356
10.1136/ANNRHEUMDIS-2015-208444
P407
P577
2016-01-18T00:00:00Z