Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway (sigmaN-sigmaS cascade) in Borrelia burgdorferi.
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Investigation of ospC Expression Variation among Borrelia burgdorferi StrainsBBA52 facilitates Borrelia burgdorferi transmission from feeding ticks to murine hosts.BmpA is a surface-exposed outer-membrane protein of Borrelia burgdorferiGenome stability of Lyme disease spirochetes: comparative genomics of Borrelia burgdorferi plasmids.CsrA modulates levels of lipoproteins and key regulators of gene expression critical for pathogenic mechanisms of Borrelia burgdorferi.Two regulatory elements required for enhancing ospA expression in Borrelia burgdorferi grown in vitro but repressing its expression during mammalian infection.Global repression of host-associated genes of the Lyme disease spirochete through post-transcriptional modulation of the alternative sigma factor RpoSOligopeptide permease A5 modulates vertebrate host-specific adaptation of Borrelia burgdorferi.Synthesis of RpoS is dependent on a putative enhancer binding protein Rrp2 in Borrelia burgdorferi.Outer surface protein OspC is an antiphagocytic factor that protects Borrelia burgdorferi from phagocytosis by macrophages.Manganese and zinc regulate virulence determinants in Borrelia burgdorferi.Contributions of environmental signals and conserved residues to the functions of carbon storage regulator A of Borrelia burgdorferi.OspC-independent infection and dissemination by host-adapted Borrelia burgdorferiOverexpression of CsrA (BB0184) alters the morphology and antigen profiles of Borrelia burgdorferi.DhhP, a cyclic di-AMP phosphodiesterase of Borrelia burgdorferi, is essential for cell growth and virulenceRegulation of the virulence determinant OspC by bbd18 on linear plasmid lp17 of Borrelia burgdorferi.Borrelia burgdorferi oxidative stress regulator BosR directly represses lipoproteins primarily expressed in the tick during mammalian infection.σ(N) -dependent control of acid resistance and the locus of enterocyte effacement in enterohemorrhagic Escherichia coli is activated by acetyl phosphate in a manner requiring flagellar regulator FlhDC and the σ(S) antagonist FliZ.Borrelia host adaptation Protein (BadP) is required for the colonization of a mammalian host by the agent of Lyme disease.
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P2860
Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway (sigmaN-sigmaS cascade) in Borrelia burgdorferi.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 October 2008
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vedecký článok
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videnskabelig artikel
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Abrogation of ospAB constituti ...... cade) in Borrelia burgdorferi.
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Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway
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Abrogation of ospAB constituti ...... cade) in Borrelia burgdorferi.
@en
Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway
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prefLabel
Abrogation of ospAB constituti ...... cade) in Borrelia burgdorferi.
@en
Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway
@nl
P2093
P2860
P1476
Abrogation of ospAB constituti ...... scade) in Borrelia burgdorferi
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P2093
Jon Blevins
Michael V Norgard
X Frank Yang
P2860
P304
P356
10.1111/J.1365-2958.2008.06491.X
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P50
P577
2008-10-23T00:00:00Z