Drak2 regulates the survival of activated T cells and is required for organ-specific autoimmune disease.
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Modulation of T Cell Metabolism and Function through Calcium SignalingDrak2 is not required for tumor surveillance and suppressionProtein kinase D orchestrates the activation of DRAK2 in response to TCR-induced Ca2+ influx and mitochondrial reactive oxygen generationSynthesis and structure-activity relationship studies of 2-(1,3,4-oxadiazole-2(3H)-thione)-3-amino-5-arylthieno[2,3-b]pyridines as inhibitors of DRAK2.Drak2 Does Not Regulate TGF-β Signaling in T Cells.Polar opposites: Erk direction of CD4 T cell subsets.Loss of DRAK2 signaling enhances allogeneic transplant survival by limiting effector and memory T cell responses.Targeting kinases for the treatment of inflammatory diseases.Regulation of the apoptosis-inducing kinase DRAK2 by cyclooxygenase-2 in colorectal cancer.
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Drak2 regulates the survival of activated T cells and is required for organ-specific autoimmune disease.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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P2093
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Drak2 regulates the survival o ...... n-specific autoimmune disease.
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Carmen Choy
Maureen A McGargill
Stephen M Hedrick
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10.4049/JIMMUNOL.181.11.7593
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P577
2008-12-01T00:00:00Z