Novel protective effects of pulsed electromagnetic field ischemia/reperfusion injury rats. - Wikidata - awgldk - TriplyDB

Novel protective effects of pulsed electromagnetic field ischemia/reperfusion injury rats.

Novel protective effects of pulsed electromagnetic field ischemia/reperfusion injury rats.

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Effects of electrical stimulation on cell proliferation and apoptosis.

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Novel protective effects of pulsed electromagnetic field ischemia/reperfusion injury rats.

http://www.wikidata.org/entity/Q37473311

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 25 October 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

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Bioscience Reports

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Novel protective effects of pu ...... hemia/reperfusion injury rats.

@en

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Aijie Zhang

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Ba Hieu Tran

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Cuilan Hou

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Fenfen Ma

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Huijuan Wang

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Rinkiko Suguro

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Ruijuan Guan

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Wenwen Li

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Xinghui Li

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YiZhun Zhu

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P2860

Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioning

Electromagnetic fields increase in vitro and in vivo angiogenesis through endothelial release of FGF-2

Pulsed electromagnetic field improves cardiac function in response to myocardial infarction.

Prosurvival Bcl-2 family members affect autophagy only indirectly, by inhibiting Bax and Bak.

Bone marrow origin of endothelial progenitor cells responsible for postnatal vasculogenesis in physiological and pathological neovascularization.

Zoledronate inhibits ischemia-induced neovascularization by impairing the mobilization and function of endothelial progenitor cells

Myocardial reperfusion injury.

Vascular Nitric Oxide: Formation and Function.

Bradykinin inhibits oxidative stress-induced cardiomyocytes senescence via regulating redox state.

Cardiac H2S Generation Is Reduced in Ageing Diabetic Mice

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scholarly article

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10.1042/BSR20160082

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6

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36

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2016-10-25T00:00:00Z

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27780890

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reperfusion injury

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5137536

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