Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1.
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Autophagy in idiopathic pulmonary fibrosisRedox mechanisms in age-related lung fibrosisMicroRNAs and oncogenic transcriptional regulatory networks controlling metabolic reprogramming in cancersReciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosisRecent developments in myofibroblast biology: paradigms for connective tissue remodelingHypoxia-Induced Epithelial-Mesenchymal Transition Is Involved in Bleomycin-Induced Lung FibrosisEpithelial-mesenchymal transition in tissue repair and fibrosisRadical-containing ultrafine particulate matter initiates epithelial-to-mesenchymal transitions in airway epithelial cellsEnvironmental particulate (PM2.5) augments stiffness-induced alveolar epithelial cell mechanoactivation of transforming growth factor betaChanges in mitochondrial function during EMT induced by TGFβ-1 in pancreatic cancerCultured alveolar epithelial cells from septic rats mimic in vivo septic lungSystems biology approaches to understanding Epithelial Mesenchymal Transition (EMT) in mucosal remodeling and signaling in asthmaHypoxia stimulates the EMT of gastric cancer cells through autocrine TGFβ signaling.Role of von Hippel-Lindau protein in fibroblast proliferation and fibrosisEpithelial-Mesenchymal Transition in tumor microenvironment.Cigarette smoke extract induces a phenotypic shift in epithelial cells; involvement of HIF1α in mesenchymal transition.Activation of hypoxia-inducible factor-1 protects airway epithelium against oxidant-induced barrier dysfunction.Hispidulin prevents hypoxia-induced epithelial-mesenchymal transition in human colon carcinoma cells.ERK/GSK3β/Snail signaling mediates radiation-induced alveolar epithelial-to-mesenchymal transition.Astaxanthin Pretreatment Attenuates Hepatic Ischemia Reperfusion-Induced Apoptosis and Autophagy via the ROS/MAPK Pathway in Mice.Angiotensinogen gene transcription in pulmonary fibrosisCombined preconditioning and postconditioning provides synergistic protection against liver ischemic reperfusion injuryProtective effect of grape seed proanthocyanidins against liver ischemic reperfusion injury: particularly in diet-induced obese mice.IL-17 induces type V collagen overexpression and EMT via TGF-β-dependent pathways in obliterative bronchiolitis.Hypoxia-inducible factor prolyl-hydroxylase-2 mediates transforming growth factor beta 1-induced epithelial-mesenchymal transition in renal tubular cells.Knockdown of von Hippel-Lindau protein decreases lung cancer cell proliferation and colonization.Suppression of von Hippel-Lindau Protein in Fibroblasts Protects against Bleomycin-Induced Pulmonary Fibrosis.The biological kinship of hypoxia with CSC and EMT and their relationship with deregulated expression of miRNAs and tumor aggressiveness.Mitochondrial retrograde signaling at the crossroads of tumor bioenergetics, genetics and epigenetics.Emodin ameliorates bleomycin-induced pulmonary fibrosis in rats by suppressing epithelial-mesenchymal transition and fibroblast activation.HIF-1α regulates EMT via the Snail and β-catenin pathways in paraquat poisoning-induced early pulmonary fibrosis.IGFBP3 promotes esophageal cancer growth by suppressing oxidative stress in hypoxic tumor microenvironmentTGF-β directs trafficking of the epithelial sodium channel ENaC which has implications for ion and fluid transport in acute lung injury.miR-17/20 Controls Prolyl Hydroxylase 2 (PHD2)/Hypoxia-Inducible Factor 1 (HIF1) to Regulate Pulmonary Artery Smooth Muscle Cell Proliferation.Hyperoxia induces alveolar epithelial-to-mesenchymal cell transition.Understanding fibrosis in systemic sclerosis: shifting paradigms, emerging opportunities.PKCδ/midkine pathway drives hypoxia-induced proliferation and differentiation of human lung epithelial cells.Fibrosis in systemic sclerosis: emerging concepts and implications for targeted therapy.Interactions between nitric oxide and hypoxia-inducible factor signaling pathways in inflammatory diseaseHIF and the lung: role of hypoxia-inducible factors in pulmonary development and disease.
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Hypoxia-induced alveolar epithelial-mesenchymal transition requires mitochondrial ROS and hypoxia-inducible factor 1.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 02 October 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@en
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@nl
type
label
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@en
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@nl
prefLabel
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@en
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@nl
P2093
P2860
P1476
Hypoxia-induced alveolar epith ...... nd hypoxia-inducible factor 1.
@en
P2093
Aileen Kelly
Guofei Zhou
Humberto Trejo
Jacob I Sznajder
John Varga
Laura A Dada
Minghua Wu
Qiyuan Zhou
P2860
P304
P356
10.1152/AJPLUNG.00007.2009
P577
2009-10-02T00:00:00Z